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Pelvic Collection | Percutaneous Catheter Drainage (Curved Needle) | 59 | Male
Pelvic Collection | Percutaneous Catheter Drainage (Curved Needle) | 59 | Male
2016advancedanteriorApriomedbeveledchibacollectioncreatedcurvaturecurvedeflectdistalfemoralgaugeglutealintroducerlateralleverlymphmarginmedialmultipleneedlepelvicposteriorposteriorlyproximalradiusreferralsacrumsciaticSIRsteerablestyletsuperiortargetingvivo
Technical Tips For Maintaining Carotid Flow During Branch Revascularization When Performing Zone 1 TEVARs
Technical Tips For Maintaining Carotid Flow During Branch Revascularization When Performing Zone 1 TEVARs
anastomosisanterioraorticarteriotomyarterybordercarotidcarotid arterycommoncreateddissectiondistalendograftflowhemostasisincisioninnominateleftlooploopsLt Subclavian RetrosmiddlepreferredprostheticproximalproximallyrestoredsecuredshuntstentsubclavianSubclavian stentsuturesystemicallyTAVRtechniquetherapeutictransversetunnelingvesselwish
Technical Tips For The Management Of Cervical And Mediastinal Iatrogenic Artery Injuries: How To Avoid Disasters
Technical Tips For The Management Of Cervical And Mediastinal Iatrogenic Artery Injuries: How To Avoid Disasters
9F Sheath in Lt SCAAbbottaccessarterybrachialcarotidcatheterCordisDual Access (Rt Femora + SC sheath) ttt with suture mediated proglid over 0.035 inch wireendovascularfemoralfrenchgraftiatrogenicimaginginjuriesleftPer-Close suture mediated ProgliderangingsheathstentsubclaviantreatedvarietyvascularvenousvertebralVessel Closure Devicewire
Highlights And Key Messages From The UK Guidance Document On AAA Treatment
Highlights And Key Messages From The UK Guidance Document On AAA Treatment
abdominalaneurysmaneurysmsaorticcomplexcostCustomised fenestrated endograftsdiscussioneffectivenessendovascularevarhighlightinfrarenalnumerousOAAAqalyrandomizedrecommendationrepairrespectrupturedSnorkel or Chimney approaches with parallel covered stentsstakeholderssurgicalthresholdthresholds
Finish Treatment Of Acute DVT In The Lab
Finish Treatment Of Acute DVT In The Lab
6-10 F AspiraxacuteAnti-coagulants & compressing stockingaspirateCDTclinicalDescending DVT - May Turner SYndromedevicedevicesDVTfemoralfollowfrenchiliofemoralmechanicalMechanical thrombectomymulticenterpatencypatientpatientsPharmacological ThrombectomypoplitealprofundaproximalseverestentsstudysubacuteswellingsymptomssyndromethrombectomythrombolysisthrombolyticthrombusTrans-Popliteal Accesstraumatictreatedtreatmentunderlyingvein
Recorded Case: Prerecorded Percutaneous Deep Venous Arterialization: Procedure Using The LimFlow Technique
Recorded Case: Prerecorded Percutaneous Deep Venous Arterialization: Procedure Using The LimFlow Technique
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Bilateral Common Iliac Aneurysms With Endo Repair Of Complex AAAs Are Troublesome And Associated With Higher Spinal Cord Ischemia And Reintervention Rates
Bilateral Common Iliac Aneurysms With Endo Repair Of Complex AAAs Are Troublesome And Associated With Higher Spinal Cord Ischemia And Reintervention Rates
abdominalaneurysmaneurysmsaorticarteryartery aneurysmbilateralbranchedcomplexcordcoveragedemographicsembolizationextensiveF/BEVARfluoroscopyhypogastriciliaciliac arteryincidenceischemiamortalityoutcomespatientspreservationproximalreinterventionspinaltreatedtreatmenttypeunilateralversus
Present Status Of The Use Of EVAR Devices For The Treatment Of Aorto-Iliac Occlusive Disease
Present Status Of The Use Of EVAR Devices For The Treatment Of Aorto-Iliac Occlusive Disease
ABF)angioplastyaortobifemoralaortobiiliacaortoiliacarteryballoonbareCERABcovereddacrondevicedevicesEndologixendovascularevarfemoralfemorisflowgraftinghemodynamicischemiakissinglimbsoutflowpatencyprofundarestenosisrevascularizationstagnationstentingstentssuperiortasctherapeuticunibody
Recorded Case: Transluminal Tumescent Anesthesia During Angioplasty Of Severely Calcified Vessels
Recorded Case: Transluminal Tumescent Anesthesia During Angioplasty Of Severely Calcified Vessels
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4D Ultrasound Evaluation Of AAAs: What Is It; How Can It Help To Predict Growth And Rupture Rates
4D Ultrasound Evaluation Of AAAs: What Is It; How Can It Help To Predict Growth And Rupture Rates
analysisaneurysmassessmentbasedbiomechanicalcontourdatadiagnosticdistalfieldgrowthimagesimaginglimitationslongitudinalmechanicalmergednephrotoxicparametersperformperformedpredictpredictorpropertiesproximalrupturesegmentationstressultrasoundvalidateviewwall
How To Treat Labial Varices: Sclerotherapy, USG Sclerotherapy And Or Phlebectomy
How To Treat Labial Varices: Sclerotherapy, USG Sclerotherapy And Or Phlebectomy
anesthesiaanteriorcomplaintsdyspareuniahemorrhageiliacincisionincludelabialLabial Varices + Leg VVligationLocal SclerotherapymalformationpatientpelvicperforatorsperformedphlebectomypolidocanolposteriorpostpartumrefluxrefluxingsaphenofemoralsclerosclerotherapysulfatesuperficialsymptomaticsymptomstetradecylultrasoundvaricositiesveinsVeithvenogramvenousversusvulvar
New In Vitro System For Evaluating Optimal Techniques, Main And Branch Endografts, Sizing, Etc. For Ch/EVAR: How It Will Improve Results And Decrease Complications By Leading To Standardized Techniques And Endograft Components
New In Vitro System For Evaluating Optimal Techniques, Main And Branch Endografts, Sizing, Etc. For Ch/EVAR: How It Will Improve Results And Decrease Complications By Leading To Standardized Techniques And Endograft Components
abdominalable covered stent (Advanta V12anatomyaneurysmanterioraorticballoonBalloon Expballoon expandablebehaviorcentersCHchimneyCordiscovereddevicedevicesendovascularEndurantevaluateevarexpandableexpansionforcegraftheterogeneityicastimpactMaquetMedtronicmitralniceoversizingpatencypercentposteriorproximalradialrenalscanstentStent graftstent graft systemStent graft system (INCRAFTstentssuboptimaltechniquetherapeuticversusviabahn
Value Of CO2 DSA For Abdominal And Pelvic Trauma: Why And How To Use CO2 Angiography With Massive Bleeding And When To Supplement It With Iodinated Contrast
Value Of CO2 DSA For Abdominal And Pelvic Trauma: Why And How To Use CO2 Angiography With Massive Bleeding And When To Supplement It With Iodinated Contrast
abdominalangiographyanterioraortaaorticarteriogrambasicallybleedingcarboncatheterceliaccoilcontrastdiaphragmdioxideembolizationholeimaginginjectinjectioninjectionsiodinatedliverlowmultiplepatientpelvicrenalruptureselectivesolublesplenictraumavascularizationveinvesselvesselsvolumes
Vascular Injuries From Orthopedic Operations: How To Prevent Catastrophes: Beware The Dangers Of Orthopedic Cement: What Are They
Vascular Injuries From Orthopedic Operations: How To Prevent Catastrophes: Beware The Dangers Of Orthopedic Cement: What Are They
acuteanterioraortaarterycementchroniccommonlycompresseddelayedfractureiliacimaginginflammatoryinjuriesinjuryinstrumentationpatientpositioningposteriorprivilegepronereplacementRt Iliac Massthermalthoracicvascularveinveinsvertebral
How To Treat By EVAR Complex Aorto-Iliac AAAs In Patients With Renal Transplants, Horseshoe Or Pelvic Kidneys: Technical Tips
How To Treat By EVAR Complex Aorto-Iliac AAAs In Patients With Renal Transplants, Horseshoe Or Pelvic Kidneys: Technical Tips
accessoryaneurysmalaneurysmsantegradeaorticapproacharteriesarteryatypicalbifurcationbypasscontralateraldistalembolizationendoendograftingendovascularevarfairlyfemoralfenestratedflowfollowuphybridhypogastriciliacincisionmaintainmaneuversmultipleocclusiveOpen Hybridoptionspatientspelvicreconstructionreconstructionsreinterventionsrenalrenal arteryrenalsrepairsurvival
Does The ATTRACT Trial Result Change How You Manage Patients With Acute DVT
Does The ATTRACT Trial Result Change How You Manage Patients With Acute DVT
abstractacuteAnti-coagulantsanticoagulationattractclotclotsdistalDVTendovascularendovascular Clot RemovalextremityfemoralinterventionpatientspharmaphlegmasiaproximalrandomizedsymptomssyndromeulcerationsveinVeithvenous
Long-Term Results Of Carotid Subclavian Bypasses In Conjunction With TEVAR: Complications And How To Avoid Them
Long-Term Results Of Carotid Subclavian Bypasses In Conjunction With TEVAR: Complications And How To Avoid Them
anteriorarterybypasscarotidcervicalcirculationcomparisoncomplicationscordcoronarydiaphragmdysfunctionendovasculargraftlandingleftLSCAnerveoriginoutcomespatencypatientsperfusionphrenicposteriorproximalpseudoaneurysmsptferesolvedrevascularizationreviewrisksspinalstentstudysubclaviansupraclavicularTEVARtherapeuticthoracicundergoingvascularvertebral
Status Of Aortic Endografts For Occlusive Disease: Indications, Precautions, Technical Tips And Value
Status Of Aortic Endografts For Occlusive Disease: Indications, Precautions, Technical Tips And Value
abisaccessacuteAFX ProthesisantegradeanterioraortaaorticaortoiliacarteriogramarteryaxillaryballoonbrachialcalcifiedcannulationcircumferentialcutdowndilatordiseasedistallyendarterectomyEndo-graftendograftendograftsEndologixexcluderExcluder Prothesis (W.L.Gore)expandableextremityfemoralfemoral arterygraftiliacintimallesionslimboccludeoccludedocclusionocclusiveOpen StentoperativeoptimizedoutflowpatencypatientspercutaneouspercutaneouslyplacementpredilationproximalrequireriskRt CFA primary repair / Lt CFA Mynx Closure devicesheathstentstentssymptomstasctechnicaltherapeuticvessels
Robotic Assisted Left Renal Vein Transposition For The Treatment Of Nutcracker Syndrome (Video)
Robotic Assisted Left Renal Vein Transposition For The Treatment Of Nutcracker Syndrome (Video)
abdominalcavaCavotomyclampclipclipscompressioncontralateraldissectionepigastricincisionsivusOpen Transposition of the Left Renal VeinovarianrenalRenal Nutcracker Syndromerenal veinretroperitoneumRobotic Assisted Open Transposition of the Renal Veinsuturetherapeutictranspositionveinvertebral
Minimally Invasive CEA Through An Incision < 3 cm In Length: Technique, Results, Precautions And Contraindications
Minimally Invasive CEA Through An Incision < 3 cm In Length: Technique, Results, Precautions And Contraindications
anterioraortobifemoralarteryatheroscleroticbifurcationcarotidcarotid arteryclassiccommondebrisemphasizeendarterectomyexternalexternal carotidfemoropoplitealhoarsenessincisioninternalinternal carotidloopmaneuvermiceminimaloperationpatientpatientsposteriorproximalpullingremovesafelyshuntsutureVeithvesselvisualizationwound
Why Open Surgery Is Still The Best Treatment For Juxta- And Pararenal AAAs In Good Risk Patients: Technical Tips
Why Open Surgery Is Still The Best Treatment For Juxta- And Pararenal AAAs In Good Risk Patients: Technical Tips
aaasadrenalanastomosisaneurysmaneurysmalaneurysmsanteriorlyaortaaorticarteryceliacclampclampingdemonstrateddissectiondivisionendovascularevarfavorablefenestratedgonadalgraftjournalleftleft renalmanipulationmedialOpen repairorificeperformedperfuseperfusedperfusionposteriorproximalproximallyrenalrenal arteryrenal veinrepairstumpsurgerysuturetechnicaltipstreatmenturetervascularveinvisceral
Pediatric Brachial Artery Injury From Supracondylar Fractures Of The Humerus: Aggressive Revascularization Is Sometimes Necessary: Indications, Technical Tips And Results
Pediatric Brachial Artery Injury From Supracondylar Fractures Of The Humerus: Aggressive Revascularization Is Sometimes Necessary: Indications, Technical Tips And Results
anteriorarterialarterybasilicbrachialcoexistingcollateralscompartmentdelayeddopplerduplexexplorationfracturefracturesfunctionhumerusinjuryinovaischemiaischemicmediannerveneurovascularnormalobservationpalpablepatientperfusedperfusionpositiveproximalpulsepulselessradialrecommendsurgicalsyndromethrombectomyvascularVeith
Treatment Of Recurrent NTOS: Role Of The Pectoralis Minor: Tips And Tricks In Diagnosis And Treatment
Treatment Of Recurrent NTOS: Role Of The Pectoralis Minor: Tips And Tricks In Diagnosis And Treatment
anteriorAnterior Axillary approach for PM Tenotomyaxialaxillarycentimeterdefinedfewerfrequentimprovementincisionindividualsinducinginjectisolatedmaneuversminormuscleneckneurogenicneurovascularoccurspalpationpatientspersistentposteriorprovocativerecurrentsymptomssyndrometendernesstypicallyulnar
2-Year ANCHOR Registry Data Show That Endoanchors Can Prevent Neck Dilatation After EVAR With Self-Expanding Devices
2-Year ANCHOR Registry Data Show That Endoanchors Can Prevent Neck Dilatation After EVAR With Self-Expanding Devices
aneurysmaortaaorticappositionarterydegenerativediameterdilatationdistalEndoanchorEndoanchor systemendograftendoleaksendovascularevargraftHeli-FXlowestMedtronicnecknegligibleoccurredoversizepatientspredictorspresenceprotectiveproximalrenalrenal arterystabilizesuprarenaltherapeuticType 1A endoleackvariables
Fully Percutaneous EVAR For RAAAs: Advantages, Contraindications And Technical Tips
Fully Percutaneous EVAR For RAAAs: Advantages, Contraindications And Technical Tips
abdominalaccessanesthesiaaneurysmaneurysmsaorticarterybaselinecolorconventionalcREVARcutdowndecreasedendovascularevarexposurefemoralimagingincreasedlengthMicropuncture TechniqueneedleoperativeoutcomespatientspercutaneousPEVARplacingregardrepairrepairsREVARRumel mediated Techniquerupturedsheathstatisticallysurgicalsuturestargettechniqueunderwentutilizing
Retrograde BTK Micro-Stenting: Initial Experience
Retrograde BTK Micro-Stenting: Initial Experience
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Lymphatic Intervention: Thoracic Duct Embolization, Pulmonary Lymphatic Perfusion Syndrome, And Hepatic Lymphatic Disorders
Lymphatic Intervention: Thoracic Duct Embolization, Pulmonary Lymphatic Perfusion Syndrome, And Hepatic Lymphatic Disorders
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Inari CloTriever Device For Acute DVT
Inari CloTriever Device For Acute DVT
anteriorbonecatheterclotCloTriever CatheterCloTriever ProcedureCloTriever SheathcompressibleCorpectomy with interbody Cage / Local Bone Graft with Local Bone PowderduplexenrollextravasationfemoralhardwareiliacinsertedLumbar Interbody fusion Via Anteriro approachlyticmaterialobstructedorthopedicoutcomespatientpatientsphasicpoplitealregistrysegmentsheathspondylolisthesisSpondylolisthesis L5-S1 / Post- Operat Acute extensive Lt Lower Limb DVTstentsubclavianswellingtherapythrombectomythrombosedthrombustibialtpaveinvenous
Expanding The Role Of The OTS t-Branch Multibranched Device From Cook For Treating TAAAs Endovascularly
Expanding The Role Of The OTS t-Branch Multibranched Device From Cook For Treating TAAAs Endovascularly
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With Adjuncts (Chimney Grafts And Onyx) 100% Of RAAAs Can Be Treated By EVAR With Low 30-Day Mortality And Turn Down Rates: Technical Tips
With Adjuncts (Chimney Grafts And Onyx) 100% Of RAAAs Can Be Treated By EVAR With Low 30-Day Mortality And Turn Down Rates: Technical Tips
abdominaladjunctsanesthesiaaorticBEVARBEVAR / Selective Gutter embolizationbrachcatheterchimneyChimney grafts with sealing below SMAcoagulationcohortcontraindicationsdeploydeployeddistalevarextendfavorableiliaclandingMedtronicmortalityNon adhesive liquid embolic agentocclusiononyxOnyx EmbolizationparallelpatientpatientsPercutaneous combined PG EVARperfusionproximalrupturedsealsealingsuprarenalSuprerenal Aneurysm / rPRAAtherapeuticthomastreatedtriplevisceral
DEBATE: Not So: Many TAAAs Are Best Treated By Open Surgery: Technical Pearls To Improve Results
DEBATE: Not So: Many TAAAs Are Best Treated By Open Surgery: Technical Pearls To Improve Results
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Transcript

who had a recent radical cystectomy and bilateral pelvic lymph node dissection. They all just created Struder neobladder seven days prior referral to us.

And he's presenting with fever and pelvic pain. His exam is only significant for a low grade fever. He got a CT scan just prior to referral and you can see fluid collection medial to the left lateral pelvic sidewall. As we dream up or think of different approaches into that collection we can see the internal lateral aspect is limited by the common femoral vessels, anteriorly

we have multiple loops of bowel. Posteriorly he had a sciatic nerve, superior gluteal artery. Obviously in this kind of collection we generally wanna stick along the lateral margin of the sacrum but even then we may actually transgress and pass the collection. At this point we thought maybe we

could use a curve needle. A curve needle is generally very useful if the target is difficult to access collections. There have been multiple papers written about targeting collections as well as targeting legions for tissue sampling. In terms of the curve needle approach,

it's benefit is that it circumvents these intervening structures which really shouldn't be transgressed. When placed coaxially it can compensate for introducer malplacement. To create one, we generally take our 22 gauge Chiba needle and take the distal 2 to 3 cm of it and curve it roughly 20 to 40 degrees.

When a curve needle is used without an introducer, it's important to remember that you have to account for that radius of curvature cuz it will be persistent throughout the advancement of the needle. Wanna introduce this new product, it's created by AprioMed. It's called the Morrison Steerable Needle. It's produced

by a company AprioMed which is out of Sweden and it comes in one size, 21 gauge and it's shaft is about 17 cm. It allows you to kind of incrementally increase the rotation of curve of your distal tip. And so as you pull down on the lever, the distal 4 cm of that needle will begin to turn and the radius of curvature begins

at about 4 cm proximal to that tip in order to deflect up to 1 cm based on how much the lever you're depressing. And so here you can see the needle being advanced from an anterior approach kinda hooking around the right femoral head and sampling a lesion posterior to the greater trochanter. I guess my one problem with this image is why they didn't just didn't

take a straight posterior approach, I don't know. But it makes for a good image and I think they tried to crop it out anyway for us. Nonetheless, ex vivo here's what you're looking at. So here is the needle, it's non beveled tip, 21 gauge, 17 cm.

And here is the inner stylet, and when you depress the stylet you do get a curvature here, again 4 cm to the distal tip. And so how much resistance does this provide to kind of tamper how much of the radius of curvature you then lose. Well when you put the two in together you do lose a little bit, and

when we measured it with a handy approach factor ex vivo we found that it was about a 20 degree deflection that you obtained. So we decided to use it for this case. We entered along the lateral margin of the sacrum, again avoiding the superior gluteal artery and sciatic nerve, but if we were to continue advancing that needle

we would miss along the medial margin of the collection. So we deflected the lever to maximum projection and we created a 20 degree kinda arc radius here and we advanced it and sure enough, the needle did advance along the expected path. And we were able to successfully drain this,

- Thank you. Here are my disclosures. Our preferred method for zone one TAVR has evolved to a carotid/carotid transposition and left subclavian retro-sandwich. The technique begins with a low transverse collar incision. The incision is deepened through the platysma

and subplatysmal flaps are then elevated. The dissection is continued along the anterior border of the sternocleidomastoid entering the carotid sheath anteromedial to the jugular vein. The common carotid artery is exposed

and controlled with a vessel loop. (mumbling) The exposure's repeated for the left common carotid artery and extended as far proximal to the omohyoid muscle as possible. A retropharyngeal plane is created using blunt dissection

along the anterior border of the cervical vertebra. A tunneling clamp is then utilized to preserve the plane with umbilical tape. Additional vessel loops are placed in the distal and mid right common carotid artery and the patient is systemically anticoagulated.

The proximal and distal vessel loops are tightened and a transverse arteriotomy is created between the middle and distal vessel loops. A flexible shunt is inserted and initially secured with the proximal and middle vessel loops. (whistling)

It is then advanced beyond the proximal vessel loop and secured into that position. The left common carotid artery is then clamped proximally and distally, suture ligated, clipped and then transected. (mumbling)

The proximal end is then brought through the retropharyngeal tunnel. - [Surgeon] It's found to have (mumbles). - An end-to-side carotid anastomosis is then created between the proximal and middle vessel loops. If preferred the right carotid arteriotomy

can be made ovoid with scissors or a punch to provide a better shape match with the recipient vessel. The complete anastomosis is back-bled and carefully flushed out the distal right carotid arteriotomy.

Flow is then restored to the left carotid artery, I mean to the right carotid artery or to the left carotid artery by tightening the middle vessel loop and loosening the proximal vessel loop. The shunt can then be removed

and the right common carotid artery safely clamped distal to the transposition. The distal arteriotomy is then closed in standard fashion and flow is restored to the right common carotid artery. This technique avoids a prosthetic graft

and the retropharyngeal space while maintaining flow in at least one carotid system at all times. Once, and here's a view of the vessels, once hemostasis is assured the platysma is reapproximated with a running suture followed by a subcuticular stitch

for an excellent cosmetic result. Our preferred method for left subclavian preservation is the retro-sandwich technique which involves deploying an initial endograft just distal to the left subclavian followed by both proximal aortic extension

and a left subclavian covered stent in parallel fashion. We prefer this configuration because it provides a second source of cerebral blood flow independent of the innominate artery

and maintains ready access to the renovisceral vessels if further aortic intervention is required in the future. Thank you.

- These are my disclosures. So central venous access is frequently employed throughout the world for a variety of purposes. These catheters range anywhere between seven and 11 French sheaths. And it's recognized, even in the best case scenario, that there are iatrogenic arterial injuries

that can occur, ranging between three to 5%. And even a smaller proportion of patients will present after complications from access with either a pseudoaneurysm, fistula formation, dissection, or distal embolization. In thinking about these, as you see these as consultations

on your service, our thoughts are to think about it in four primary things. Number one is the anatomic location, and I think imaging is very helpful. This is a vas cath in the carotid artery. The second is th

how long the device has been dwelling in the carotid or the subclavian circulation. Assessment for thrombus around the catheter, and then obviously the size of the hole and the size of the catheter.

Several years ago we undertook a retrospective review and looked at this, and we looked at all carotid, subclavian, and innominate iatrogenic injuries, and we excluded all the injuries that were treated, that were manifest early and treated with just manual compression.

It's a small cohort of patients, we had 12 cases. Eight were treated with a variety of endovascular techniques and four were treated with open surgery. So, to illustrate our approach, I thought what I would do is just show you four cases on how we treated some of these types of problems.

The first one is a 75 year-old gentleman who's three days status post a coronary bypass graft with a LIMA graft to his LAD. He had a cordis catheter in his chest on the left side, which was discovered to be in the left subclavian artery as opposed to the vein.

So this nine French sheath, this is the imaging showing where the entry site is, just underneath the clavicle. You can see the vertebral and the IMA are both patent. And this is an angiogram from a catheter with which was placed in the femoral artery at the time that we were going to take care of this

with a four French catheter. For this case, we had duel access, so we had access from the groin with a sheath and a wire in place in case we needed to treat this from below. Then from above, we rewired the cordis catheter,

placed a suture-mediated closure device, sutured it down, left the wire in place, and shot this angiogram, which you can see very clearly has now taken care of the bleeding site. There's some pinching here after the wire was removed,

this abated without any difficulty. Second case is a 26 year-old woman with a diagnosis of vascular EDS. She presented to the operating room for a small bowel obstruction. Anesthesia has tried to attempt to put a central venous

catheter access in there. There unfortunately was an injury to the right subclavian vein. After she recovered from her operation, on cross sectional imaging you can see that she has this large pseudoaneurysm

coming from the subclavian artery on this axial cut and also on the sagittal view. Because she's a vascular EDS patient, we did this open brachial approach. We placed a stent graft across the area of injury to exclude the aneurism.

And you can see that there's still some filling in this region here. And it appeared to be coming from the internal mammary artery. We gave her a few days, it still was patent. Cross-sectional imaging confirmed this,

and so this was eventually treated with thoracoscopic clipping and resolved flow into the aneurism. The next case is a little bit more complicated. This is an 80 year-old woman with polycythemia vera who had a plasmapheresis catheter,

nine French sheath placed on the left subclavian artery which was diagnosed five days post procedure when she presented with a posterior circulation stroke. As you can see on the imaging, her vertebral's open, her mammary's open, she has this catheter in the significant clot

in this region. To manage this, again, we did duel access. So right femoral approach, left brachial approach. We placed the filter element in the vertebral artery. Balloon occlusion of the subclavian, and then a stent graft coverage of the area

and took the plasmapheresis catheter out and then suction embolectomy. And then the last case is a 47 year-old woman who had an attempted right subclavian vein access and it was known that she had a pulsatile mass in the supraclavicular fossa.

Was noted to have a 3cm subclavian artery pseudoaneurysm. Very broad base, short neck, and we elected to treat this with open surgical technique. So I think as you see these consults, the things to factor in to your management decision are: number one, the location.

Number two, the complication of whether it's thrombus, pseudoaneurysm, or fistula. It's very important to identify whether there is pericatheter thrombus. There's a variety of techniques available for treatment, ranging from manual compression,

endovascular techniques, and open repair. I think the primary point here is the prevention with ultrasound guidance is very important when placing these catheters. Thank you. (clapping)

- Thank you very much for the opportunity and there are obviously certain areas of overlap with the SVS guidelines. These are my conflicts of interest. Firstly, I'd like to pose to you people two questions. Do you believe in evidence based medicine? And do you think cost is important?

These are the core principles of how NICE works. It likes to obtain the best evidence and will only consider Level I RCT evidence. There's expert input. There's patient and carer involvement and it is completely open and transparent procedure.

There are many things that go into making a recommendation. But, the key thing is based on cost effectiveness. If we also look at the SVS guidelines, obviously cost effectiveness is actually quite important. Two of their research recommendations highlight the importance of cost effectiveness.

But, the main thing that they highlight is that if you have a constrained resource, you need to do a cost effectiveness analysis as to how you will actually manage patients. So, is cost effectiveness important? Well, the American view is it definitely is.

So, with the NICE guidance, a topic is identified, there are numerous stakeholders, then a committee is appointed, questions and agendas are set, reviewed by the stakeholders, then the work begins, then a draft document goes out for consultation

and a final guideline is published. And there are numerous stakeholders and these are an example of the stakeholders in the United Kingdom with respect to the Vascular Society, BSET, British Society for Interventional Radiology, industry and everybody had their say

before the questions were set. If we then look at the NICE guidance group, there are obviously experts, there are patients, there's a whole NICE team that consist of anything up to 16 people and I got involved when Matt Thompson resigned

and I took over Matt's place. Now, I'm going to talk to you about the draft guidelines. The final guidelines are going to be released on December, 19th. Now, I'm just going to highlight some important issues. It's really quite clear that in the UK it was felt

that you should treat asymptomatic aneurysms at 5.5, if they were symptomatic or if there was more than a one centimeter growth in one year if they were 4 centimeters. But, the other key recommendation is do not offer endovascular aneurysm repair

to an unruptured infrarenal abdominal aortic aneurysm if open surgical repair is suitable. And this is quite important and it is also important that it is determined by their anesthetic and medical condition. Similarly, do not offer complex EVAR

to patients with an unruptured abdominal aortic aneurysm if open repair is possible other than if there is a randomized control trial. Now, complex aneurysms for the purpose of this are basically defined as an aneurysm that could not be treated by an infrarenal device

within it's IFU or anything extending anywhere up to the diaphragm and we can have a discussion and argument, but this is how a complex aneurysm was defined at the original committee meeting. Now, I can see you'll all think that this is

going to be a little bit bizarre because for repairing ruptured aneurysms the recommendation is that you should consider, and consider is a mild recommendation from NICE that you should consider offering an EVAR, particularly, to women and men over the age of 70.

But, very clearly, do not offer complex EVAR to people with a ruptured abdominal aortic aneurysm if open surgical repair is suitable, again, other than in part of a randomized control trial. There was some discussion and this caused quite a lot of heated discussion about monitoring for

and after endovascular repair. And you should read this wording very carefully. It says do not use color duplex ultrasound as the main imaging technique to detect endoleaks in people who have an EVAR. It doesn't mention anything about looking for sac expansion.

There were recommendations with respect for research, one, similar to the SVS about frequency and thresholds and a question with respect to complex aneurysms that we need in RCT. Now, were any of these results anything unexpected?

Well, I would suggest to you not. If you look at all of the literature, nobody other than for the OVER trial has ever shown that endovascular aneurysm repair is cost effective. If you then look at the UK cost effectiveness that was done by NICE, open repair dominated EVAR.

I you looked at EVAR-2 i.e. not fit for open repair, to gain one QALY cost 450,000. Whereas the threshold in the UK is 20,000. So, I would suggest to you that any independent, unbiased committee could not have recommended to the NHS to purchase EVAR over open repair.

So, ladies and gentlemen, I would you to think that how you spend your money in the 21st century. But, I would also commend to you, we have only done what the SVS have recommended, that you should be judging treatment on cost effectiveness. But, also remember that the US thresholds

are significantly different for the value of a QALY and look how much difference we spend on national GDP, 8% compared to 18%. So, ladies and gentlemen, these are our draft documents. The official ones will be released later on this month, next month,

but they are based on Level I evidence looking at a cost effectiveness threshold and I completely agree that there are many areas of controversy. Thank you very much.

- You already heard about different devices which can finish the treatment of acute DVT in the lab and I would like to add one of the devices which is quite widespread in Europe. And share the first study on this device. This is called the Aspirex device. So what is the objective?

Post traumatic syndrome after proximal DVT, I think that's clear. 25% of the patient are at risk for developing post traumatic syndrome. I think that is clear and some of these patient even expect severe post traumatic syndrome.

We already saw this ATTRACT trial outcome and we learned that especially patient with Iliofemoral DVT might benefit from treatment, invasive treatment of Iliofemoral DVT but of course, we need to know that is catheter-directed thrombolysis causes issues

and therefore our way should be to go away from thrombolytic therapy to a pure mechanical thrombectomy approach. This is a typical case example of a patient, 20 year old female patient who came to the emergency room with that leg on the left side in the morning,

back pain in the evening and this is clear that it is a descending Iliofemoral DVT in that patient caused by May-Thurner syndrome. So, with modern devices like this Aspirex, mechanical thrombectomy device, the 10 French device is able to aspirate up to 130 millimeter,

ml per minute of clots. You see that this can be effectively treated and then stinted within the May-Thurner syndrome within one session approach. So, but, what is clear of course that we need to get data

for these modern Mechanical Thrombectomy devices and therefore, we conducted clinical follow-up study to evaluate safety and efficiency of that Aspirex Mechanical Thrombectomy device. This device is based on the Archimedic principle which you can see here it comes with six up

to 10 French systems and with that you are able, as I already showed to sac 130ml of thrombus per minute. So these are the study details I want to show you. We treated 50 psychs, 56 patients with acute, subacute and acute on chronic which means up to 3 months of symptoms patients with Iliofermal DVT.

We performed IVIS on all these patients. We found May-Thurner syndrome in at least half of these patients as a reason for the Iliofermal DVT. You see the patient demographics. Some of the patients had even malignancy condition. A lot of patients were on oral contraceptives.

Here are the clinical symptoms within our cohort. Most of the patients came with swelling and rest pain. The rVCSS at the beginning was 4.5 within this cohort. Most of the traumatic lesions were on the left side involving even the profunda and the common femoral vein in this cohort.

You see here the excess which we used for treating these Iliofermal DVT, we used in the main part of the cohort, the left popliteal vein access or left femoral vein access. 84% were treated with 10 French system, the Aspirex device. As I mentioned we used IVIS

to analyze underlying pathologies. We found in most of the patients underlying pathologies and this explains why we implanted stents in 100% of the patients. You see the treatment duration which was in mean 94 minutes within this treatment cohort.

These are the patency analysis within one year. You see patency at 12 months, 87% percent in these patients, which we could follow up after 12 months. Here you see the Post-thrombotic syndrome analysis after 12 months so only low PTS

and some kind of moderate PTS were seen in these patients. There were no severe Post-thrombotic syndrome. Most of the patients just had a little bit of swelling after that procedure. Of course, it's important to mention safety and those end points.

There were just some small punctures associated, site being complicationS. Of course re-hospitalization is a severe adverse event which you can see here. But there were of course no bleeding events in this cohort. And to follow up

on this much more multicentric perspective trial, we just started a multicenter trial on this and we'll follow up patients up to five years within this just initiated multicenter registry. And I think we can show some preliminary data next year. Thank you very much.

- So, this is a prerecorded case. This is my disclosures. Well, we've all seen the concept of Percutaneous Venous Arterialization and this was discussed several sessions ago in the main hall of going crossing from an artery into vein and having multiple stent step would re-line the

venous wall and take away the valves and to bring blood to the capillary bit. Quickly, this is the animation of what goes on. An antegrade seven french sheath, a retrograde five french sheath. An ultrasonic system with alignment between the

artery and the vein and then a needle is driven, wired, pre-dilatation. The use of a 4 french valvulotome to cut the valves in the foot, to de-valvulotomize the valves and then after that, align the whole vein with a covered stent

to do an endovenous bypass. So, this is a prerecorded case courtesy of Dr. Daniel van Den Heuvel, an Interventional Radiologist out of Netherlands. So, this is a 70 year old lady with ulcer in the first and the fifth toe, Rutherford five.

Diabetic, hypertensive and failed conventional intervention. So, I'll start the video right now. Could we have volume please? Yeah, thank you. - Okay. (beeping) - Feel free to ask any questions.

I can pause the video. - [Doctor] So, we see proximal occlusions of the tibial vessels, and all collaterals going down to the foot. So, no direct target vessel for revascularization. So we apply the tourniquet with an Esmarch.

- So now, he's actually getting venous access at the-- - (mumbles) in the vessel. - Retro (mumbles) area. - Gently past the probe. Okay, so now the-- - I'll just pause over here. This is the device with the retrograde five french sheath

and the five french catheter, and the antegrade device that allows you to cross over an artery to the vein, and this is properly aligned in this case. - [Doctor] Marker of the arterial probe is at the same level as the venous probe. Let's fix it with a wet gauze, please.

And then, let's fix it and connect the arterial probe. So, I think this is the maximal-- - So, the ultrasound system allows a predictable crossing between an artery and the vein. - [Doctor] Feed and retract the venous probe. So, leave the sheath in while they are about

to retract the venous probe. Maybe, yeah. Also, pull down the sheath a little bit. - After alignment, the one probe is removed to give space for the crossing. - [Doctor] Yup, okay.

To puncture with a needle, you need to turn the device to unlock it and then advance it. Yeah. So, I unlock it. Okay. Yeah, this, see some contrast.

So, at the same time, we'll do the puncture with the needle. - That was quite subtle. You could see the needle push against the vein wall and it went right through the vein wall. - [Doctor] And then see what happens. - And, now, with an over the wire system,

you're able to introduce a wire. - [Doctor] It's in right away. - You know you're in the vein because of the appearance of the wire abutting a valve. - [Doctor] We can try to correct for that in the later stage.

- So, now, with a support of a CXI catheter, he's trying to catheterize the appropriate vein. - This is probably the right (mumbles) so. Okay, so this is the right one, so we crossed in the middle.

So, now, again, we go distal and lead gently. Try to, aha! Now, we're in the sheath. Okay, false.

Try for several times. If not, then-- So, okay, so probably now, already past the puncture point. Be gentle, just wait for the valve to open and not to push too much.

- So, the next step is to cross the valves and this maneuver is usually done with a variety of wires and the main thing that you need is actually patience. - Don't go too far (mumbles)-- - So, this could be supported with a balloon or a CXI catheter or any other catheter. - Yeah, okay.

Okay, and the wire is better. But, is it the right one? Yeah, okay. - The tourniquet is still on that you have gauzes or is it-- - Usually, at this point, after the crossing, you can actually take the tourniquet off, so--

- [Professor] The contrast is staying here in the-- - Yes, I think he probably injected some contrast through a superficial vein to maybe give a better visualization of the anatomy. So, I think, in this case, probably there was some either the tonic,

it wasn't off, or he injected some contrast. It can be useful. You know, it's not harmful to the patient. - [Doctor] Vein we're targeting, so. Now, we should be able to cross the-- - So, there are plenty of valves

in the foot. - The feet arch. - There's usually one in the mid area here. So, he was able to cross this. And then, after that, you're able to cross the venous arch with no problems because it's not diseased as like the arterial arch.

So, fairly predictable pattern of, very much similar the arterial system is in this case. - [Doctor] So, now, you open up the valvulotome. Close the two E again. Now, you can do like forward cutting with the valvulotome

It has, you see, and we know where there are valves here. You see the tapering of the valvulotome and now it's open. - So, when the valvulotome engages a valve, maybe I can double back a little bit. - [Doctor] Now it's open.

- A little bit more. - [Doctor] Forward cutting with the valvulotome. - So, when the valvulotome engages the valve, you can see a nearing of the valvulotome and then suddenly there's a jump and then the valve, the valvulotome opens up again.

- [Doctor] The valvulotome and now its open. - That's it. So, that's the typical appearance of how a valvulotome would cut the valve and we would rotate the valvulotome and cut it in multiple locations.

And then, the, this is implantation with a stent and we would generally avoid the bifurcation of the medial lateral plantar vein. So, this stent is a 5.5 millimeters at the bottom end. And, a tapered stent on the top. Maybe I'll just quickly--

- [Doctor] No more space, so I will just-- Dip finish. - Move up, so as he's continuing to stack up the stents-- - [Doctor] Preparing some, yeah. - And then, do an angiogram. You can see the area that you want to cross.

- [Doctor] So, now we can clearly see the ostium of the posterior tip. So, I want to stay distal of that. - So, this is done from a 3.5 to a 5.5 stent to make sure that we don't cover the origin of the perioral artery to prevent ex--

Occlusion of the collateral. - [Doctor] Do a longer inflation, so we have to five balloon again. - And post dilatation is done and-- - Okay. - In this case. - [Doctor] Yeah.

Four millimeter or a five. - This is done with a five. This looks like a five millimeter. Basically, to essentially rupture the vein, the vein is only serving as a conduit and after that, the stent is able to expand. So, this is a very nice picture

of good flow-- - It's nice. - No residual stenosis and good flow up the arch as well. And then, at the proximal end, I believe, he probably did some post dilatation as well. - [Doctor] Okay, here. - And sometimes, we do add on a (mumbles)

stent if the vessel is diseased on top, you know, but I think not in this case. In the proximal end, very good flow. Over here, the origin and-- So, this patient, pre-procedure, had gangrene over the tip of the toe and after that,

the gangrene had healed and on this site, not sure if this is gangrene or just a nail infection, but we know it got better and you can see that the foot is visibly different between before and after the procedure. It always look a little bit swollen,

but swelling has never become a major issue on a chronic phase. We haven't had patients come back with venous ulceration or anything along those lines. So, with that, I'd like to thank you. And this is a 3-D rotational angiogram

of, not a CT scan, rotation angiogram of what it looks like after the procedure. Thanks. - [Man] Fine. (applauding) - [Professor] Thank you, Steven,

for this great presentation, a really fascinating technology. I have a small question over-- - Yes, Professor. - What about digital arteries? Because in your picture, I never saw the digital arteries

after the bypass. - Yeah, Professor, that's a very good question and this is a problem that we continue to grapple with and we find it's seems to be unable to be solved because there are valves at the metatarsal and digital veins that go into the, to the toe.

And, one of the shortcomings of this procedure, it seems that we are unable to push the blood as distal as we want to push like a conventional angioplasty. So, I'm not sure if we're able to circumvent this for the future because, as you know,

the veins in the foot and the toes are really really very small and prone to spasm and very high restenosis. So, in the future, if we're able to bring the blood forward instead of a fly by across the foot, you know, like a conventional angiogram,

this may hold a future for what we're doing. But, until now, we don't really have something like that. What we can do and what Roberto has done is to focalize and to selectively pressurize the flow forward with a selective embolization of large collaterals like the great saphenous vein

or even surgical ligation. But, this is, you have to do it very gently 'cause if you, if you take away these outflows, then there's a risk of thrombosis, but if you have too many outflows, then the blood does not reach the foot in front,

you know, so this could be a potential for studies in future. Maybe pressure monitoring or something along that line. Yeah. - And what about the patency? - The patency?

Well, the patency is not very good in this procedure, as I must confess, you know. We do have re-intervention for this procedures, but a re-intervention is actually quite simple if you are able to survey it, mostly like a bypasses in the distal end.

So, one wire, one drug coated balloon would usually solve the problem if you detect it before thrombosis. If you do have thrombosis, then it's going to be a problem because it's like a bypass. You have to use a Rotarex disc, that's been my experience,

to clean up the whole graph. Quite quick and then do a bit of thrombolysis to clear the clot that's in the veins and certainly the secondary patency is not so good if the, if the veins all thrombose because with thrombosis,

the veins have a intense scarring and inflammatory process, so I find that the benefit of re-intervention early is much better in terms of the clinical as well as the angiographic outcome and the perfusion as well.

So, surveillance like how you would survey for a distal bypass and treat the stenosis early. - It's Lisa. (mumbles) - Okay, thank you very much for this very nice presentation.

- That's a long title, thank you. We shortened the title, and just said, The Iliac Artery's Complicating Complex Juxtarenal and Thoracal Abdominal Repair. I have no disclosures. So, Iliac artery preservation is important whenever we start doing complex aortic aneurysm repair.

We don't understand completely what the incidence is with these extensive aneurysms. We know with AAAs, anywhere in the 10 to 40% have some sort of iliac artery involvement. It certainly can complicate the management as we get to these more complicated repairs.

Iliac artery preservation may be important for prevention of spinal cord ischemia, and those people in whom we can maintain both hypogastric arteries, it occurs at a less significant rate, with less severe symptoms and higher rates of recovery.

The aim of our study was to evaluate the incidence, management, and outcomes of iliac artery aneurysms associated with complex aortic aneurysms treated with fenestrated and branched endografts. Part of a PS-IDE study over a 15 year period of time,

this is dated from the Cleveland Clinic for the treatment of juxtarenal aneurysms and thoracal abdominal aortic aneurysms. For the purpose of this study, we defined an iliac artery aneurysm is 21 mm or greater as determined by diameter

by our core lab. We chose 21 mm because this was outside of the IFU for the iliac wounds that we had currently available to us at that time. We did multivariable analysis on the number of different outcomes. And we looked at the incidence

of iliac artery aneurysms by repair type. In all the aneurysms we treated, we see about a third of the patients had some level of iliac artery aneurysm involvement. In those patients that had less extensive thoracal abdominals, the type three

and type four abdominals, it occurred in about a third of the cases. A little bit less than the type two and the type one thoracal abdominals. We look at the demographics between those that had iliac artery aneurysm

involvement and those that did not have iliac artery involvement. It was more common in males to have iliac artery involvement than any other group. There are more females that didn't have iliac artery aneurysms. The rest

of the demographics were the same between the two groups. We look at the anatomic characteristics of the iliac artery aneurysms, about 60% of them were unilateral, about 40% of them were bilateral.

The mean iliac artery aneurysm size was 28 mm and that was the same on both sides. And we look at thought the percent that were actually very large, or considered large enough to potentially in and of themselves the repairs

greater than three centimeters. About 28% of them were greater than three centimeters on each side. If we look at our iliac artery aneurysm treatment type, this is 509 iliac artery aneurysms that

were treated out of all these patients. About 46% of them, we were able to obtain a seal distal to the iliac artery aneurysm. So it really only involved the proximal portion, the proximal half of the iliac artery.

20% of them, we placed a hypogastric branched endograft, and about 20% of them, we placed a hypogastric coverage plus embolization of that internal iliac artery. About 13% of them were left untreated at the time for a variety of different operative reasons.

Why is there a difference between the hypogastric coverage and embolization? It was availability of devices and surgeon choice at the time. At one point, we had a opportunity to be able to treat both fairly easily

on both sides and at one point we did not. Larger iliac artery aneurysms were treated with hypogastric coverage or hypogastric branched endografts, and there was a significant difference between the two. Most of the mean

size of those that were actually treated with either hypogastric branch or embolization for greater than three centimeters. If we look at peri-operative outcomes in those without iliac artery aneurysms versus those with iliac artery aneurysms.

We see that the fluoroscopy estimated blood loss is larger for those with iliac artery aneurysms, fluoroscopy time was longer and procedure duration was a bit longer as well. Obviously, a bit more complicated procedure,

more steps that's going to take a little bit longer to perform them. It did not effect the length of stay for these patients or the length of stay in the intensive care unit following the procedures. We look

at all-cause mortality at five years, no difference in whether they had an iliac artery aneurysm or not. It didn't matter whether it was unilateral or bilateral. If we look at aneurysm-related mortality, it's the same whether

they had the iliac artery aneurysm or not. Same for unilateral versus bilateral as well. Where we start to see some differences are the freedom from reintervention. This did vary between, among the three groups. In those patients without an iliac

artery aneurysm, they had the lower reintervention rate than those with the unilateral iliac artery aneurysm, and even lower rates from freedom from reintervention in those that had bilateral iliac artery aneurysms. Spinal cord ischemia, one of the

reasons we try to preserve both the hypogastric arteries. Look at our total spinal cord ischemia incidents. It didn't vary between the two groups, but if we look specifically, the type two thoracal abdominal aortic aneurysms in those patients that had bilateral

iliac arte higher rate of spinal cord ischemia compared to those that did not have any iliac artery aneurysms or those that had an internal iliac, a single iliac artery aneurysm.

So, iliac artery aneurysms affect about a third of the patients with complex aortic disease. They do not, their presence does not affect all-cause mortality or aneurysm related mortality. They are associated with a higher reintervention rate.

In extensive aneurysms, may be higher association with higher spinal cord ischemia rates. We need additional efforts are needed to improve outcomes and understanding appropriate application of different treatment options for patients with

complex aortic disease. Thank you.

- I have no disclosures relevant to this talk. So we're going to be talking today a little bit about some old devices with some new results, some new devices with some better results, and these are the EVAR-like devices, and you're going to hear a little bit

about open surgery in what should be primarily an endovascular talk, but I believe that everything old is new again and I hope I can convince you of the same. Now, the objectives with endovascular surgery, specifically in the aortoiliac segment,

is to get similar results to aortobifemoral bypassing or open aortoiliac surgery. So on the left you see the classic Dacron graft, of an aortobifemoral graft, and on the right the kissing balloon stents. So where have we been?

Well in 2011, and I don't need to introduce this to this crowd, the COBEST Trial, 168 randomized limbs in a one-to-one ratio. Bare metal stents to covered stents with binary restenosis being the primary outcome. They also did a sub-analysis by TASC classification,

and I think most people are aware of this data. If you look at all lesions, covered stents are superior to bare metal stents, and this is really quite pronounced when looking at TASC C and D lesions. And again, this is in the aorto-iliac segment.

So the take-home message as of 2011 is kissing balloon stents using, kissing balloon angioplasty using covered stents. So do we have a new gold standard on the block? Well, the three to five year patency of aortobifemoral bypass grafting is in the 90% range.

Kissing balloon stents, even with covered stents, really only gets you about 60 to 70% three to five years. So these are not equal. Why is this? Well the thought is there are flow disturbances

at the neo-bifurcation. Enter covered endovascular reconstruction of the aortic bifurcation, or CERAB. On the left side of the panel I think that device will be recognized by most people here as the Unibody Endologix device,

and on the right is a device designed by Dr. Goverde in Antwerp, who has published extensively on this, which is basically two V12 stents placed into a unibody. I talked a little bit about flow dynamics,

and a great paper from Jebbink. What he found was in kissing balloon angioplasty using covered stents, there's a flow mismatch. As one can see on the right side of the slide, it shows kissing stents, and the red areas represent blood flow

that is actually around the covered stents. And what he found by studying this is there's actually flow stagnation and increase in vortices in the area that are probably promotive of neo-intimal hyperplasia, and therefore not giving you the same longevity

that an aortobifemoral would give. So with CERAB, what about patency? Again, a study from Dr. Goverde's group in Antwerp in 130 patients, relatively high percentage with critical limb ischemia and about 90% having TASC D lesions.

At three years he produces a primary patency of 87% and the clinically-driven target lesion revascularization similarly at 87%. These are impressive results. It's a well-done study, and the thought being that by using a covered stent,

one can reduce that flow stagnation in the vortices that are promoting neo-intimal hyperplasia. So is this the end of the aortobifemoral graft? We've got 90% aortobifemoral, 60 to 70% with kissing balloon stents and 87% using the CERAB technique.

But I'm going to challenge everybody. Have we compared apples to apples? When you look at Goverde's study, and again it's very well-written. If you look at his outflow vessels, and again, this is the aortoiliac segment,

over 90% of his patients have a deep femoral artery that's patent. So this is actually aortobiiliac grafting as opposed to aortobifemoral grafting. And I think that we need to go back to one of our, to really what I think

is a classic paper on this, and this is from 2008, the Cleveland Clinic Group, where they had 127 limbs again, and it was a comparison in a retrospective fashion to aortobifemoral grafting and covered stenting. And what I want to bring to your attention here

is not the fact that the two procedures perform well, is that more than a quarter of these patients had an outflow procedure, and that was primarily profundoplasty. So what's new? Covered stents are certainly superior.

I think the CERAB technique is superior to kissing stents and I think that there's a hemodynamic advantage here. I think that the data is good. However, everything old is new again. I'm calling 2018 the year of the profunda. So if you're considering stenting

in the aortoiliac segment and whether that's, whatever your comfort zone is, be it kissing balloon or the CERAB technique, remember our first principles. Inflow, outflow equals blood flow. The outflow of the iliacs, by and large,

is the profunda femoris artery. So I advocate a liberal use of common femoral artery repair and I think that as the devices improve and as our experience improves, I think CERAB is here to stay and is a very promising technique.

Thank you.

- [Presenter] Thank you very much. So this is a case where, recently, we found it very convenient for our patients when we do good vessel preparation with, let's say, aggressive balloon dilatation to anaesthetize the whole artery or the whole length, we have several options open to do this and this is the recorded case where we

use here a special balloon to do this the Bullfrog Device. So this is a 76 old patient we have done this case just last week as a live case for VIVA ulceration forefoot right but also severe claudication for longer time

short walking distance, ABI low, a severely calcified SFA, total occlusion on the right side and here you can see the risk factors. So before I start this is the device which were using here for the local anesthesia of the whole artery.

The Bullfrog Device, so a balloon with a needle here inside the foldings when you open up the balloon the needle is pressed into the artery, into the adventitia or perivascular tissue and then you can inject here actually whatever you want.

It has been trialed in the DANCE trial injecting Cortisone, Dexamethasone into the artery as a phase but also in below the knee arteries. But here's now the case. Can we have volume please? - [Surgeon] In fact, it looks a little bit different to

what we thought it would be so we have seen clearly a lot of calcium but here the proximal part looks to me actually a little bit thrombotic although complains we had no acute onset. We can see the proximal part is not that calcified so I would actually call this rather severely calcified

and seeing this, I would say, well in our lab a superior case. You can however also see more distal, some more disease. This is more a stenotic and potentially, if we can we would only maybe only treat this with a drug called balloon.

The outflow is very slow and posterior at the end is the main artery is also very much to this disease and the prosterior is actually the only one it will go down. Thought it would be relatively a complex case to recannalise with wire because of this calcification. Took an O18 wire, command 18

and the wire just flew thorough. It was a little bit surprising. We want to pre-dilate now. We want to do here some kind of local anesthesia to that artery. And in a lady with this severe calcification

we always, I mean expect quite some pain when we balloon here especially when we aggressively balloon to prepare fore superior stents and we have since some years something like a standard to really take needle down here to the plaque and give local anesthesia here around that plaque to make it

completely pain free, this treatment. So at the beginning we did this with X-ray control to take the needle down then we change to ultrasound guided puncture which is also actually very nice. Well, recently that's what we want to show you here,

we actually took the Bullfrog Device to inject local anesthesia into the arterial wall and found that actually very nice. Extremely easy and we don't have to puncture the skin above that artery several times which is also of course sometimes, I would especially in female patients,

not the nicest thing to do. - [Caller] I just caught up on where you are right now and if you have concerns about thrombosis, is there a conversation you had about some embolisation protection... - [Surgeon] Pre-dilated now a little bit

also for that Bullfrog Device to get easily through this severe calcium here with 3 mm balloon which was, patient fell but of course, 3 mm was not too painful. I think that 3 mm pre-dilation may also help me to identify thrombus.

- [Presenter] This is spider filter 7 mm. - [Surgeon] Embolisation. So now I have the Bullfrog Device here to go down and as I just heard, I mean calcium could be a concern for Bullfrog Device to be capable to bring the needle through the calcium.

- [Caller] Tell us a little bit about the concentration you're using there and how you'll determine how far apart each of your injections are going to be - [Surgeon] Yeah. - [Caller] Were you just visualizing that depending on the calcification or what's the parameters

that you're using there under. - [Surgeon] Well, it's actually as we have done it in the trials that you mix what you want to inject here into the arterial wall with some contrast. So, if we have 2% Lidocaine, 20 mm plus 4 mm of contrast and that enables me to really see whether

the needle is going into the arterial wall or whether the injection is into the artery. So we're going to inflate now, the balloon. You can see those two markers here. We'll go little close here now, which is now two markers direct.

Maybe you can also see that small needle here getting out. Just a moment. I think you can see that small needle if it's not calcium. Just a moment. Okay, now I load here maybe.. - [Caller] So this is, what 30, 34 gauge needle? - [Surgeon] Yes. Okay, let's inject here a little bit.

And as you can see here, it goes into the surrounding. I think quite nicely and you see it's spreads also proximal and distal and that means I don't, I can deflate now of course and to an area where I do not see it repull. Well the repull was not too strong infact.

That's here, please. - [Caller] Now is she having any pain with this pull of inflation or we'll she notice some of the earlier? - [Surgeon] Absolutely no sensation here of that balloon inflation.

Let's see. So this here, I think again goes very nicely into the arterial wall, as you can see it spreads distally a little bit. In fact, my first case I did with Bullfrog in the SFA was a case where there was actually no calcium but still extremely painful for the patient

that of course worked very nicely. And the second case however I had a real, extremely calcified SFA which was however in fact very eccentric long occlusion and the wire of course went subintimal or down beside the plaque, between adventitia which was not calcified in the plaque

and it was very easy to direct the needle to the non-calcified area and even though it was very calcified, it was very easy to give a successful administration of local into every area. So, here of course, it's a little bit different. This is now a circumferential calcification,

wire went through intraluminally. - [Caller] So, you're starting with you're intention to - [Presenter] So let me stop here. Just quickly. So maybe, Steven, there's several ways to do local anesthesia to the whole artery. I know you're doing something different.

Michael, for example, he injects some Lidocaine into the artery. I found it sometimes helpful but it may make pain little better but they are not completely pain free. You're doing some local injection there around the femoral nerve in the groin.

Can you report how this works? - [Steven] Yup, so Andrea you're right. I think as we all know, the strategy behind calcified lesion is adequate vessel preparations. So, the way we do it is to inject the mixture of Lidocaine as well as the Bupivacaine,

which is the long acting anesthesia. So, mixture of short acting and long acting and ultra-sound guidance, we look for the femoral nerve which is lateral to the artery and we infuse a 20 to 30 mm volume around the femoral nerve and for the BTK intervention 100% of all our BTK interventions we do a nerve block

at the popliteal level to completely anesthetized the leg below the knee and this allows high pressure angioplasty 24, 26 atmospheres with completely no pain at all. And also no movement because it blocks the sensory and also the motor nerve and the patients recover

with no sequelae. It's quite a common practice for orthopedic surgical procedures so and so forth. But I think the concept of putting a Lidocaine into the adventitial space even within the arterial wall is a very very interesting concept for when other techniques of anesthesia is not available.

I think the Bullfrog Device, you can see it spread. I think it's not, maybe the areas where you inject and it goes, I think it's actually a possibly that adventitia is diffusing very nicely with anesthesia. And I'm just interested that if the patient had any pain at all cause I think in this cause...

- [Presenter] Nothing, no sensation anymore at all. - [Steven] No sensation.. completely. So, I mean do you do it on one side? Two sides? - [Presenter] Actually, only on one side. You have seen it on one injection, it spreads around - [Steven] Spreads around right?

- [Presenter] But it also goes up and down. - [Steven] Yeah. I mean, I'm just curious just looking at this procedure that calcified vessel maybe before the anesthesia, the needle will also help to change the compliance of the calcium by puncturing - [Presenter] Potentially. That requires,

however, severally punctures. (Steven laughs) Let me continue here the case to the end. Just a moment. Hopefully it works. - [Caller] Maybe the stent size you're going to use. Maybe go to six...

- [Surgeon] I do in fact, in this extremely calcified arteries in men often go because it's absolutely pain free to a 6.5. However, this lady here, I think it's rather small arteries unfortunately, I think the maximum would be 5.5. The middle portion, I had the impression that maybe

balloon inflation was not complete, the balloon opening was not complete and the severe calcium would in fact go - [Presenter] So, six, 40, 20 atmospheres have no sensation at all. Nothing. - [Surgeon] I think we can come here without stents

so therefore, I continue here... - Why you done today use this drug a called balloon? - [Presenter] Distal, you use drug called balloon, but more proximal not. Yes, I started right here where the occlusion ended with the superior stent.

- [Surgeon] We didn't have any problem here to open up the stent here and I think it really looks very regular. Here the calcium, here actually no problem here, had to push the device a little bit in here to stay regular with this stent and here it went out.

Just to show you here that really nearly nominal a little bit stacked when you take the delivery system down, the markers here indicate 150 mm. - [Caller] When I get one that looks that good I'd take the risk... (Surgeon laughs)

I mean this is something what you wouldn't do in studies and there's no limitation of trials but might be at the end of the day - [Presenter] Drug-eluting Eluvia stent proximal - [Caller] ...Which is best stent? Or because you use the areas where the mechanical

requirements are most important and optimize the stent like Supera. And to use the arrowverse scaffolding is important but let's say you don't use heavy calcification drug-eluting device which probably gives you good results for many other reasons

and makes the procedure just easier and precise so at the end of the day it's very meaningful combination in my eye. - [Presenter] Yeah, fine. Thank you. There's some few slices. This is how we started with percutaneous local anesthesia

and the X-ray with a longer needle to go down to the plaque and give it at several areas. Here you can another angulation how the needle goes down to the plaque and we change to ultrasound which is also very nice, no X-ray can see also better how it spreads here, you know where to

puncture the next time to give another local anesthesia. Just two more cases where we used the Bullfrog which were actually nicer than in this case here. Here you can see that really with every injection here you could deliver a nice depull. This case we were doing that was very old lady

with a very fragile artery. I think this was problem. But other cases where really absolutely easy to do this here. Once again the needle which goes in here and again 6 mm balloon. No sensation of pain whereas predilation with 4 mm balloon was extremely painful in this patient.

And then we thought it can not work in calcium because in calcific arteries below the knee we sometimes had problems to get the needle out of the calcium into the adventitia. Here it's different usually it's wire travel eccentrically around the plaque and you can direct this needle here

to other side, not to the calcium but to the other side. So also with extremely calcium where we actually need this most here for, let's say for crack and pave technique, it worked actually very nice here with all the depulls here with a nice result. Thank you.

- Thank you. I'm grateful. It's a great presentation. (Crowd clapping) This is really an innovative idea and for me the best application of the Bullfrog balloon. (Speaker laughs) - [Presenter] But also you could maybe mix it

with local something else... - Because my next question is, are you injecting Lidocaine and Dexamethasone simultaneously? - [Presenter] Haven't done it yet but that would be... - This could be a good idea that you inject Lidocaine and Dexamethasone simultaneously.

- Thank you very much. After these beautiful two presentations a 4D ultrasound, it might look very old-fashioned to you. These are my disclosures. Last year, I presented on 4D ultrasound and the way how it can assess wall stress. Now, we know that from a biomechanical point,

it's clear that an aneurysm will rupture when the mechanical stress exceeds the local strength. So, it's important to know something about the state of the aortic wall, the mechanical properties and the stress that's all combined in the wall.

And that could be a better predictor for growth and potential rupture of the aneurysm. It has been performed peak wall stress analysis, using finite element analysis based on CT scan. Now, there has been a test looking at CT scans with and without rupture and given indication

what wall stress could predict in growth and rupture. Unfortunately, there has been no longitudinal studies to validate this system because of the limitations in radiation and nephrotoxic contrast. So, we thought that we could overcome these problems and building the possibilities for longitudinal studies

to do this similar assessment using ultrasound. As you can see here in this diagram in CT scan, mechanical properties and the wall thickness is fixed data based on the literature. Whereas with 3D ultrasound, you can get these mechanical properties from patient-specific imaging

that could give a more patient-specific mechanical AA model. We're still performing a longitudinal study. We started almost four years ago. We're following 320 patients, and every time when they come in surveillance, we perform a 3D ultrasound. I presented last year that we are able to,

with 3D ultrasound, we get adequate anatomy and the geometry is comparable to CT scan, and we get adequate wall stressors and mechanical parameters if we compare it with CT scan. Now, there are still some limitations in 3D ultrasound and that's the limited field of view and the cumbersome procedure and time-consuming procedures

to perform all the segmentation. So last year, we worked on increased field of view and automatic segmentation. As you can see, this is a single image where the aneurysm fits perfectly well in the field of view. But, when the aneurysm is larger, it will not fit

in a single view and you need multi-perspective imaging with multiple images that should be fused and so create one image in all. First, we perform the segmentation of the proximal and distal segment, and that's a segmentation algorithm that is

based on a well-established active deformable contour that was published in 1988 by Kass. Now, this is actually what we're doing. We're taking the proximal segment of the aneurysm. We're taking the distal segment. We perform the segmentation based on the algorithms,

and when we have the two images, we do a registration, sort of a merging of these imaging, first based on the central line. And then afterwards, there is an optimalisation of these images so that they finally perfectly fit on each other.

Once we've done that, we merge these data and we get the merged ultrasound data of a much larger field of view. And after that, we perform the final segmentation, as you can see here. By doing that, we have an increased field of view and we have an automatic segmentation system

that makes the procedure's analysis much and much less time-consuming. We validate it with CT scan and you can see that on the geometry, we have on the single assessment and the multi assessments, we have good similarity images. We also performed a verification on wall stress

and you can see that with these merged images, compared to CT scan, we get very good wall stress assessment compared to CT scan. Now, this is our view to the future. We believe that in a couple of years, we have all the algorithms aligned so that we can perform

a 3D ultrasound of the aorta, and we can see that based on the mechanical parameters that aneurysm is safe, or is maybe at risk, or as you see, when it's red, there is indication for surgery. This is where we want to go.

I give you a short sneak preview that we performed. We started the analysis of a longitudinal study and we're looking at if we could predict growth and rupture. As you can see on the left side, you see that we're looking at the wall stresses. There is no increase in wall stress in the patient

before the aneurysm ruptures. On the other side, there is a clear change in the stiffness of the aneurysm before it ruptures. So, it might be that wall stress is not a predictor for growth and rupture, but that mechanical parameters, like aneurysm stiffness, is a much better predictor.

But we hope to present on that more solid data next year. Thank you very much.

- Like to thank Dr. Veith and the committee for asking me to speak. I have no conflicts related to this presentation. Labial and vulvar varicosities occur in up to 10% of pregnant women, with the worst symptoms being manifested in the second half of the pregnancy.

Symptoms include genital pressure and fullness, pruritus, and a sensation of prolapse. These generally worsen with standing. Management is usually conservative. Between compression hose, cooling packs, and exercise, most women can make it through to the end of the pregnancy.

When should we do more than just reassure these women? An ultrasound should be performed when there's an early presentation, meaning in the first trimester, as this can be an unmasking of a venous malformation. If there are unilateral varicosities,

an ultrasound should be performed to make sure that these aren't due to iliac vein thrombosis. If there's superficial thrombosis or phlebitis, you may need to rule out deep venous extension with an ultrasound. When should we intervene?

You may need to intervene to release trapped blood in phlebitis, or to give low molecular weight heparin for comfort. When should a local phlebectomy or sclerotherapy be performed? Should sclerotherapy be performed during pregnancy?

We know very little. Occasionally, this is performed in a patient who is unknowingly pregnant, and there have been no clear complications from this in the literature. The effectiveness of sclero may also

be diminished in pregnancy, due to hormones and increased venous volume. Both polidocanol and sodium tetradecyl sulfate say that there is no support for use during pregnancies, and they advise against it. So what should you do?

This following case is a 24 year old G2P1, who was referred to me at 24 weeks for disabling vaginal and pelvic discomfort. She couldn't go to work, she couldn't take care of her toddler, she had some left leg complaints, but it was mostly genital discomfort and fullness,

and her OB said that he was going to do a pre-term C-section because he was worried about the risk of hemorrhage with the delivery. So this is her laying supine pre-op, and this is her left leg with varicosities visible in the anterior and posterior aspects.

Her ultrasound showed open iliac veins and large refluxing varicosities in the left vulvar area. She had no venous malformation or clot, and she had reflux in the saphenofemoral junction and down the GSV. I performed a phlebectomy on her,

and started with an ultrasound mapping of her superficial veins and perforators in the labial region. I made small incision with dissection and tie ligation of all the varicosities and perforators, and this was done under local anesthesia

with minimal sedation in the operating room. This resulted in vastly improved comfort, and her anxiety, and her OB's anxiety were both decreased, and she went on to a successful delivery. So this diagram shows the usual location of the labial perforators.

Here she is pre-op, and then here she is a week post-op. Well, what about postpartum varicosities? These can be associated with pelvic congestion, and the complaints can often be split into local, meaning surface complaints, versus pelvic complaints.

And this leads into a debate between a top down treatment approach, where you go in and do a venogram and internal coiling, versus a bottom up approach, where you start with local therapy, such as phlebectomy or sclero.

Pelvic symptoms include aching and pressure in the pelvis. These are usually worse with menstruation, and dyspareunia is most pronounced after intercourse, approximately an hour to several hours later. Surface complaints include vulvar itching, tenderness, recurrent thrombophlebitis, or bleeding.

Dyspareunia is present during or at initiation of sexual intercourse. I refer to this as the Gibson Algorithm, as Kathy Gibson and I have talked about this problem a lot, and this is how we both feel that these problems should be addressed.

If you have an asymptomatic or minimally symptomatic patient who's referred for varicosities that are seen incidentally, such as during a laparoscopy, those I don't treat. If you have a symptomatic patient who has pelvic symptoms, then these people get a venogram with coils and sclerotherapy as appropriate.

If they are not pregnant, and have no pelvic symptoms, these patients get sclero. If they are pregnant, and have no pelvic symptoms, they get a phlebectomy. In conclusion, vulvar varicosities are a common problem, and usually conservative management is adequate.

With extreme symptoms, phlebectomy has been successful. Pregnancy-related varicosities typically resolve post-delivery, and these can then be treated with local sclerotherapy if they persist. Central imaging and treatment is successful for primarily pelvic complaints or persistent symptoms.

Thank you.

- Thank you very much for the nice introduction for the privilege to start the aortic session with this nice, very interesting topic about Chimney technique and especially about the in-vitro testing which we have done in Muenster in Germany. So, the Chimney endovascular technique we treat short necks as we see here.

With the use of off-the-shelf devices and the placement is in parallel and outside configuration of the main abdominal device. Well, if you see the literature we can see enthusiastic reports with the use of these alternative therapeutic options,

showing low incidence of endo leaks, excellent patency, and durability of these endovascular solutions. On the other side we have also centers with suboptimal experience, as we see here from Manchester, in the titled already publication,

late ruptures after single chimneys or from the group from Florida, highlight that the technique raises cause for concern. So what are the reasons for these divergent experience? Could be the heterogeneity of the used materials, but also the degree of oversizing

of the aortic stent graft? In order to evaluate that, we performed first of all a chimney case of a patient with a huge paraanastomotic aneurysm which we did with single chimney for the right renal artery, as we see here.

What we have done is the CT scan of these patients, we send to a special company and create this silicone model one by one with the anatomy of this treated case, as we see here, having a diameter of 28 millimeter, exact the anatomy of the renals,

of the neck length, infrarenally. And it was also really nice the opportunity to have a fluid simulation system, and we can have also the possibility to bring the device in the CT scan, and perform CT angiography, as we can see here,

very nicely the pictail catheter into the descending aorta, and evaluating now the impact of the different devices for this technique. Here is the example with the device you see here how we deploy the chimney graft, here is an Incraft stent graft for the right renal artery.

The first attempt was to evaluate the impact of different abdominal devices. If we use the same chimney graft in this particular case, the Icast for Advanta V12, and you see what we changed was only the type of the stent graft of the aorta.

If you see here the CT scan analysis, you see very nicely these combination of a mitral endoskeleton of the enduring device with a rigid, but very good, intraradial force Advanta V12, or Icast. You can see here how nicely performs

around the chimney graft. And if we see also in the reconstruction, we have a very nice expansion of the chimney graft, especially in the proximal edge, which is very important in order to have a good patency over the time.

You see here very nicely the expansion of the proximal edge of the balloon expandable covered stent Let's see now what happened with the Incraft. Again, you see here very nice the radial force of the Icast is here very nice to see. However, we have seen a completely different behavior

of the abdominal stent graft of this company. You see here that we have potentially more gutters compared to the other conformability of the endurant around the chimney graft. So it was a very nice sign and finding and showing the impact of the abdominal stent graft

for this technique. What we have done after that was we took the endurant device and we changed now the use of the chimney graft, so we used in the first attempt the self-expanding covered stents, the Viabahn,

versus a balloon expandable covered stent like the Begraft. And if you see here the results, you see again a very nice expansion of the endurant around the chimney graft, but in the reconstruction you see here the severe compression of the Viabahn self-expanding stent

has poor radial force despite that we lined we had per se 70 percent stenosis. I think it's a very important finding crucial compared to the balloon expandable chimney grafts you see here the Begraft, they had also a very good expansion

as balloon-expandable covered did, but also we see here completely different area of gutters if we compare the two balloon expandable covered stents in the anterior and posterior phase, you see here the Begraft plus seems to perform better. The impact of degree of oversizing we know

from the work of Riambaud 30 percent is the recommendation if you see this very nice analysis, you see here with 15 percent oversizing, we have this area of gutter versus 30 percent of oversizing you see a very nice conformability around the chimney that we chose how important is for this technique

to have enough fabric material to wrap up the chimney grafts. In conclusion, ladies and gentlemen, we have seen in this very nice in-vitro testing that indeed the area of the gutters vary depending on the different device combinations.

And also we have seen how important is the appropriate device selection, and 30 percent oversizing to obtain optimal results. Thank you very much.

- Thank you very much for the opportunity to speak carbon dioxide angiography, which is one of my favorite topics and today I will like to talk to you about the value of CO2 angiography for abdominal and pelvic trauma and why and how to use carbon dioxide angiography with massive bleeding and when to supplement CO2 with iodinated contrast.

Disclosures, none. The value of CO2 angiography, what are the advantages perhaps? Carbon dioxide is non-allergic and non-nephrotoxic contrast agent, meaning CO2 is the only proven safe contrast in patients with a contrast allergy and the renal failure.

Carbon dioxide is very highly soluble (20 to 30 times more soluble than oxygen). It's very low viscosity, which is a very unique physical property that you can take advantage of it in doing angiography and CO2 is 1/400 iodinated contrast in viscosity.

Because of low viscosity, now we can use smaller catheter, like a micro-catheter, coaxially to the angiogram using end hole catheter. You do not need five hole catheter such as Pigtail. Also, because of low viscosity, you can detect bleeding much more efficiently.

It demonstrates to the aneurysm and arteriovenous fistula. The other interesting part of the CO2 when you inject in the vessel the CO2 basically refluxes back so you can see the more central vessel. In other words, when you inject contrast, you see only forward vessel, whereas when you inject CO2,

you do a pass with not only peripheral vessels and also see more central vessels. So basically you see the vessels around the lesions and you can use unlimited volumes of CO2 if you separate two to three minutes because CO2 is exhaled by the respirations

so basically you can inject large volumes particularly when you have long prolonged procedures, and most importantly, CO2 is very inexpensive. Where there are basically two methods that will deliver CO2. One is the plastic bag system which you basically fill up with a CO2 tank three times and then empty three times

and keep the fourth time and then you connect to the delivery system and basically closest inject for DSA. The other devices, the CO2mmander with the angio assist, which I saw in the booth outside. That's FDA approved for CO2 injections and is very convenient to use.

It's called CO2mmander. So, most of the CO2 angios can be done with end hole catheter. So basically you eliminate the need for pigtail. You can use any of these cobra catheters, shepherd hook and the Simmons.

If you look at this image in the Levitor study with vascular model, when you inject end hole catheter when the CO2 exits from the tip of catheter, it forms very homogenous bolus, displaces the blood because you're imaging the blood vessel by displacing blood with contrast is mixed with blood, therefore as CO2

travels distally it maintains the CO2 density whereas contrast dilutes and lose the densities. So we recommend end hole catheter. So that means you can do an arteriogram with end hole catheter and then do a select arteriogram. You don't need to replace the pigtail

for selective injection following your aortographies. Here's the basic techniques: Now when you do CO2 angiogram, trauma patient, abdominal/pelvic traumas, start with CO2 aortography. You'll be surprised, you'll see many of those bleeding on aortogram, and also you can repeat, if necessary,

with CO2 at the multiple different levels like, celiac, renal, or aortic bifurcation but be sure to inject below diaphragm. Do not go above diaphragm, for example, thoracic aorta coronary, and brachial, and the subclavian if you inject CO2, you'll have some serious problems.

So stay below the diaphragm as an arterial contrast. Selective injection iodinated contrast for a road map. We like to do super selective arteriogram for embolization et cetera. Then use a contrast to get anomalies. Super selective injection with iodinated contrast

before embolization if there's no bleeding then repeat with CO2 because of low viscocity and also explosion of the gas you will often see the bleeding. That makes it more comfortable before embolization. Here is a splenic trauma patient.

CO2 is injected into the aorta at the level of the celiac access. Now you see the extra vascularization from the low polar spleen, then you catheterize celiac access of the veins. You microcatheter in the distal splenic arteries

and inject the contrast. Oops, there's no bleeding. Make you very uncomfortable for embolizations. We always like to see the actual vascularization before place particle or coils. At that time you can inject CO2 and you can see

actual vascularization and make you more comfortable before embolization. You can inject CO2, the selective injection like in here in a patient with the splenic trauma. The celiac injection of CO2 shows the growth, laceration splenic with extra vascularization with the gas.

There's multiple small, little collection. We call this Starry Night by Van Gogh. That means malpighian marginal sinus with stagnation with the CO2 gives multiple globular appearance of the stars called Starry Night.

You can see the early filling of the portal vein because of disruption of the intrasplenic microvascular structures. Now you see the splenic vein. Normally, you shouldn't see splenic vein while following CO2 injections.

This is a case of the liver traumas. Because the liver is a little more anterior the celiac that is coming off of the anterior aspect of the aorta, therefore, CO2 likes to go there because of buoyancy so we take advantage of buoyancy. Now you see the rupture here in this liver

with following the aortic injections then you inject contrast in the celiac axis to get road map so you can travel through this torus anatomy for embolizations for the road map for with contrast. This patient with elaston loss

with ruptured venal arteries, massive bleeding from many renal rupture with retro peritoneal bleeding with CO2 and aortic injection and then you inject contrast into renal artery and coil embolization but I think the stent is very dangerous in a patient with elaston loss.

We want to really separate the renal artery. Then you're basically at the mercy of the bleeding. So we like a very soft coil but basically coil the entire renal arteries. That was done. - Thank you very much.

- Time is over already? - Yeah. - Oh, OK. Let's finish up. Arteriogram and we inject CO2 contrast twice. Here's the final conclusions.

CO2 is a valuable imaging modality for abdominal and pelvic trauma. Start with CO2 aortography, if indicated. Repeat injections at multiple levels below diaphragm and selective injection road map with contrast. The last advice fo

t air contamination during the CO2 angiograms. Thank you.

- I'd like to thank Dr. Veith and the committee for the privilege of presenting this. I have no disclosures. Vascular problems and the type of injuries could be varied. We all need to have an awareness of acute and chronic injuries,

whether they're traumatic, resulting with compression, occlusion, tumoral and malformation results, or vasospastic. I'd like to present a thoracoscopic manipulation of fractured ribs to prevent descending aortic injury

in a patient with chest trauma. You know, we don't think about this but they can have acute or delayed onset of symptoms and the patient can change and suddenly deteriorate with position changes or with mechanical ventilation,

and this is a rather interesting paper. Here you can see the posterior rib fracture sitting directly adjacent to the aorta like a knife. You can imagine the catastrophic consequences if that wasn't recognized and treated appropriately.

We heard this morning in the venous session that the veins change positions based on the arteries. Well, we need to remember that the arteries and the whole vascular bundle changes position based on the spine

and the bony pieces around them. This is especially too when you're dealing with scoliosis and scoliotic operations and the body positioning whether it's supine or prone the degree of hypo or hyperkyphosis

and the vertebral angles and the methods of instrumentation all need to be considered and remembered as the aorta will migrate based on the body habits of the patient. Screws can cause all kinds of trouble.

Screws are considered risky if they're within one to three millimeters of the aorta or adjacent tissues, and if you just do a random review up to 15% of screws that are placed fall into this category.

Vertebral loops and tortuosity is either a congenital or acquired anomaly and the V2 segment of the vertebral is particularly at risk, most commonly in women in their fifth and sixth decades,

and here you can see instrumentation of the upper cervical spine, anterior corpectomy and the posterior exposures are all associated with a significant and lethal, at times, vertebral artery injuries.

Left subclavian artery injury from excessively long thoracic pedicle screws placed for proximal thoracic scoliosis have been reported. Clavicular osteosynthesis with high neurovascular injury especially when the plunge depth isn't kept in mind

in the medial clavicle have been reported and an awareness and an ability to anticipate injury by looking at the safe zone and finding this on the femur

with your preoperative imaging is a way to help prevent those kinds of problems. Injuries can be from stretch or retraction. Leave it to the French. There's a paper from 2011 that describes midline anterior approach

from the right side to the lumbar spine, interbody fusion and total disc replacement as safer. The cava is more resistant to injury than the left iliac vein and there's less erectile dysfunction reported. We had a patient present recently

with the blue bumps across her abdomen many years after hip complicated course. She'd had what was thought to be an infected hip that was replaced, worsening lower extremity edema, asymmetry of her femoral vein on duplex

and her heterogeneous mask that you can see here on imaging. The iliac veins were occluded and compressed and you could see in the bottom right the varicosities that she was concerned about. Another case is a 71-year-old male who had a post-thrombotic syndrome.

It was worsened after his left hip replacement and his wife said he's just not been the same since. Initially imaging suggests that this was a mass and a tumor. He underwent biopsy

and it showed ghost cells. Here you can see the venogram where we tried to recanalize this and we were unsuccessful because this was actually a combination of bone cement and inflammatory reaction.

Second patient in this category, bless you, is a 67-year-old female who had left leg swelling again after a total hip replacement 20 plus years ago. No DVTs but here you can see the cement compressing the iliac vein.

She had about a 40% patency when you put her through positioning and elected not to have anything done with that. Here you could see on MR how truly compressed this is. IVA suggested it was a little less tight than that.

So a vascular injury occurs across all surgical specialties. All procedures carry risk of bleeding and inadvertent damage to vessels. The mechanisms include tearing, stretching, fracture of calcific plaques,

direct penetration and thermal injury. The types of injuries you hear are most common after hip injuries, they need to be recognized in the acute phase as looking for signs of bleeding or ischemia. Arterial lesions are commonly prone then.

Bone cement can cause thermal injury, erosion, compression and post-implant syndrome. So again, no surgery is immune. You need to be aware and especially when you look at patients in the delayed time period

to consider something called particle disease. This has actually been described in the orthopedic literature starting in the 70s and it's a complex interaction of inflammatory pathways directed at microparticles that come about

through prosthetic wear. So not only acute injury but acute and chronic symptoms. Thank you for the privilege of the floor.

- Good morning, thank you, Dr. Veith, for the invitation. My disclosures. So, renal artery anomalies, fairly rare. Renal ectopia and fusion, leading to horseshoe kidneys or pelvic kidneys, are fairly rare, in less than one percent of the population. Renal transplants, that is patients with existing

renal transplants who develop aneurysms, clearly these are patients who are 10 to 20 or more years beyond their initial transplantation, or maybe an increasing number of patients that are developing aneurysms and are treated. All of these involve a renal artery origin that is

near the aortic bifurcation or into the iliac arteries, making potential repair options limited. So this is a personal, clinical series, over an eight year span, when I was at the University of South Florida & Tampa, that's 18 patients, nine renal transplants, six congenital

pelvic kidneys, three horseshoe kidneys, with varied aorto-iliac aneurysmal pathologies, it leaves half of these patients have iliac artery pathologies on top of their aortic aneurysms, or in place of the making repair options fairly difficult. Over half of the patients had renal insufficiency

and renal protective maneuvers were used in all patients in this trial with those measures listed on the slide. All of these were elective cases, all were technically successful, with a fair amount of followup afterward. The reconstruction priorities or goals of the operation are to maintain blood flow to that atypical kidney,

except in circumstances where there were multiple renal arteries, and then a small accessory renal artery would be covered with a potential endovascular solution, and to exclude the aneurysms with adequate fixation lengths. So, in this experience, we were able, I was able to treat eight of the 18 patients with a fairly straightforward

endovascular solution, aorto-biiliac or aorto-aortic endografts. There were four patients all requiring open reconstructions without any obvious endovascular or hybrid options, but I'd like to focus on these hybrid options, several of these, an endohybrid approach using aorto-iliac

endografts, cross femoral bypass in some form of iliac embolization with an attempt to try to maintain flow to hypogastric arteries and maintain antegrade flow into that pelvic atypical renal artery, and a open hybrid approach where a renal artery can be transposed, and endografting a solution can be utilized.

The overall outcomes, fairly poor survival of these patients with a 50% survival at approximately two years, but there were no aortic related mortalities, all the renal artery reconstructions were patented last followup by Duplex or CT imaging. No aneurysms ruptures or aortic reinterventions or open

conversions were needed. So, focus specifically in a treatment algorithm, here in this complex group of patients, I think if the atypical renal artery comes off distal aorta, you have several treatment options. Most of these are going to be open, but if it is a small

accessory with multiple renal arteries, such as in certain cases of horseshoe kidneys, you may be able to get away with an endovascular approach with coverage of those small accessory arteries, an open hybrid approach which we utilized in a single case in the series with open transposition through a limited

incision from the distal aorta down to the distal iliac, and then actually a fenestrated endovascular repair of his complex aneurysm. Finally, an open approach, where direct aorto-ilio-femoral reconstruction with a bypass and reimplantation of that renal artery was done,

but in the patients with atypical renals off the iliac segment, I think you utilizing these endohybrid options can come up with some creative solutions, and utilize, if there is some common iliac occlusive disease or aneurysmal disease, you can maintain antegrade flow into these renal arteries from the pelvis

and utilize cross femoral bypass and contralateral occlusions. So, good options with AUIs, with an endohybrid approach in these difficult patients. Thank you.

- Thank you to the moderators, thank you to Dr. Veith for having me. Let's go! So my topic is to kind of introduce the ATTRACT trial, and to talk a little bit about how it affected, at least my practice, when it comes to patients with acute DVT.

I'm on the scientific advisory board for a company that makes IVC filters, and I also advise to BTG, so you guys can ask me about it later if you want. So let's talk about a case. A 50-year-old man presents

from an outside hospital to our center with left lower extremity swelling. And this is what somebody looks like upon presentation. And pulses, motor function, and sensation are actually normal at this point.

And he says to us, "Well, symptoms started "three days ago. "They're about the same since they started," despite being on anticoagulation. And he said, "Listen guys, in the other hospital, "they wouldn't do anything.

"And I want a procedure because I want the clot "out of me." so he's found to have this common femoral vein DVT. And the question is should endovascular clot removal be performed for this patient?

Well the ATTRACT trial set off to try and prevent a complication you obviously all know about, called the post-thrombotic syndrome, which is a spectrum from sort of mild discomfort and a little bit of dyspigmentation and up

to venous ulcerations and quite a lot of morbidity. And in ATTRACT, patients with proximal DVT were randomized to anticoagulation alone or in combination with pharma mechanical catheter-directed thrombolysis.

And the reason I put proximal in quotes is because it wasn't only common sort of femoral vein clots, but also femoral vein clots including the distal femoral vein were included eventually. And so patients with clots were recruited,

and as I said, they were randomized to those two treatments. And what this here shows you is the division into the two groups. Now I know this is a little small, but I'll try and kind of highlight a few things

that are relevant to this talk. So if you just read the abstract of the ATTRACT trial published last year in the New England Journal of Medicine, it'll seem to you that the study was a negative study.

The conclusion and the abstract is basically that post-thrombotic syndrome was not prevented by performing these procedures. Definitely post-thrombotic syndrome is still frequent despite treatment. But there was a signal for less severe

post-thrombotic syndrome and for more bleeding. And I was hoping to bring you all, there's an upcoming publication in circulation, hopefully it'll be online, I guess, over the weekend or early next week, talking specifically about patients

with proximal DVT. But you know, I'm speaking now without those slides. So what I can basically show you here, that at 24 months, unfortunately, there was no, well not unfortunately,

but the fact is, it did cross the significance and it was not significant from that standpoint. And what you can see here, is sort of a continuous metric of post-thrombotic syndrome. And here there was a little bit of an advantage

towards reduction of severe post-thrombotic syndrome with the procedure. What it also shows you here in this rectangle, is that were more bleeds, obviously, in the patients who received the more aggressive therapy.

One thing that people don't always talk about is that we treat our patients for two reasons, right? We want to prevent post-thrombotic syndrome but obviously, we want to help them acutely. And so what the study also showed,

was that acute symptoms resolved more quickly in patients who received the more aggressive therapy as opposed to those who did not. Again, at the price of more bleeding. So what happened to this patient? Well you know,

he presented on a Friday, obviously. So we kind of said, "Yeah, we probably are able "to try and do something for you, "but let's wait until Monday." And by Monday, his leg looked like this, with sort of a little bit of bedrest

and continued anticoagulation. So at the end of the day, no procedure was done for this particular patient. What are my take home messages, for whatever that's worth? Well I think intervention for DVT

has several acute indications. Restore arterial flow when phlegmasia is the problem, and reduce acute symptoms. I think intervention for common femoral and more proximal DVT likely does have long-term benefit, and again, just be

on the lookout for that circ paper that's coming out. Intervention for femoral DVT, so more distal DVT, in my opinion, is rarely indicated. And in the absence of phlegmasia, for me, thigh swelling is a good marker for a need

for a procedure, and I owe Dr. Bob Schainfeld that little tidbit. So thank you very much for listening.

- Our group has looked at the outcomes of patients undergoing carotid-subclavian bypass in the setting of thoracic endovascular repair. These are my obligatory disclosures, none of which are relevant to this study. By way of introduction, coverage of the left subclavian artery origin

is required in 10-50% of patients undergoing TEVAR, to achieve an adequate proximal landing zone. The left subclavian artery may contribute to critical vascular beds in addition to the left upper extremity, including the posterior cerebral circulation,

the coronary circulation if a LIMA graft is present, and the spinal cord, via vertebral collaterals. Therefore the potential risks of inadequate left subclavian perfusion include not only arm ischemia, but also posterior circulation stroke,

spinal cord ischemia, and coronary insufficiency. Although these risks are of low frequency, the SVS as early as 2010 published guidelines advocating a policy of liberal left subclavian revascularization during TEVAR

requiring left subclavian origin coverage. Until recently, the only approved way to maintain perfusion of the left subclavian artery during TEVAR, with a zone 2 or more proximal landing zone, was a cervical bypass or transposition procedure. As thoracic side-branch devices become more available,

we thought it might be useful to review our experience with cervical bypass for comparison with these newer endovascular strategies. This study was a retrospective review of our aortic disease database, and identified 112 out of 579 TEVARs

that had undergone carotid subclavian bypass. We used the standard operative technique, through a short, supraclavicular incision, the subclavian arteries exposed by division of the anterior scalene muscle, and a short 8 millimeter PTFE graft is placed

between the common carotid and the subclavian arteries, usually contemporaneous with the TEVAR procedure. The most important finding of this review regarded phrenic nerve dysfunction. To exam this, all pre- and post-TEVAR chest x-rays were reviewed for evidence of diaphragm elevation.

The study population was typical for patients undergoing TEVAR. The most frequent indication for bypass was for spinal cord protection, and nearly 80% of cases were elective. We found that 25 % of patients had some evidence

of phrenic nerve dysfunction, though many resolved over time. Other nerve injury and vascular graft complications occurred with much less frequency. This slide illustrates the grading of diaphragm elevation into mild and severe categories,

and notes that over half of the injuries did resolve over time. Vascular complications were rare, and usually treated with a corrective endovascular procedure. Of three graft occlusions, only one required repeat bypass.

Two pseudoaneurysms were treated endovascularly. Actuarial graft, primary graft patency, was 97% after five years. In summary then, the report examines early and late outcomes for carotid subclavian bypass, in the setting of TEVAR. We found an unexpectedly high rate

of phrenic nerve dysfunction postoperatively, although over half resolved spontaneously. There was a very low incidence of vascular complications, and a high long-term patency rate. We suggest that this study may provide a benchmark for comparison

with emerging branch thoracic endovascular devices. Thank you.

- Thank you for asking me to speak. Thank you Dr Veith. I have no disclosures. I'm going to start with a quick case again of a 70 year old female presented with right lower extremity rest pain and non-healing wound at the right first toe

and left lower extremity claudication. She had non-palpable femoral and distal pulses, her ABIs were calcified but she had decreased wave forms. Prior anterior gram showed the following extensive aortoiliac occlusive disease due to the small size we went ahead and did a CT scan and confirmed.

She had a very small aorta measuring 14 millimeters in outer diameter and circumferential calcium of her aorta as well as proximal common iliac arteries. Due to this we treated her with a right common femoral artery cutdown and an antegrade approach to her SFA occlusion with a stent.

We then converted the sheath to a retrograde approach, place a percutaneous left common femoral artery access and then placed an Endologix AFX device with a 23 millimeter main body at the aortic bifurcation. We then ballooned both the aorta and iliac arteries and then placed bilateral balloon expandable

kissing iliac stents to stent the outflow. Here is our pre, intra, and post operative films. She did well. Her rest pain resolved, her first toe amputation healed, we followed her for about 10 months. She also has an AV access and had a left arterial steel

on a left upper extremity so last week I was able to undergo repeat arteriogram and this is at 10 months out. We can see that he stent remains open with good flow and no evidence of in stent stenosis. There's very little literature about using endografts for occlusive disease.

Van Haren looked at 10 patients with TASC-D lesions that were felt to be high risk for aorta bifem using the Endologix AFX device. And noted 100% technical success rate. Eight patients did require additional stent placements. There was 100% resolution of the symptoms

with improved ABIs bilaterally. At 40 months follow up there's a primary patency rate of 80% and secondary of 100% with one acute limb occlusion. Zander et all, using the Excluder prothesis, looked at 14 high risk patients for aorta bifem with TASC-C and D lesions of the aorta.

Similarly they noted 100% technical success. Nine patients required additional stenting, all patients had resolution of their symptoms and improvement of their ABIs. At 62 months follow up they noted a primary patency rate of 85% and secondary of 100

with two acute limb occlusions. The indications for this procedure in general are symptomatic patient with a TASC C or D lesion that's felt to either be a high operative risk for aorta bifem or have a significantly calcified aorta where clamping would be difficult as we saw in our patient.

These patients are usually being considered for axillary bifemoral bypass. Some technical tips. Access can be done percutaneously through a cutdown. I do recommend a cutdown if there's femoral disease so you can preform a femoral endarterectomy and

profundaplasty at the same time. Brachial access is also an alternative option. Due to the small size and disease vessels, graft placement may be difficult and may require predilation with either the endograft sheath dilator or high-pressure balloon.

In calcified vessels you may need to place covered stents in order to pass the graft to avoid rupture. Due to the poor radial force of endografts, the graft must be ballooned after placement with either an aortic occlusion balloon but usually high-pressure balloons are needed.

It usually also needs to be reinforced the outflow with either self-expanding or balloon expandable stents to prevent limb occlusion. Some precautions. If the vessels are calcified and tortuous again there may be difficult graft delivery.

In patients with occluded vessels standard techniques for crossing can be used, however will require pre-dilation before endograft positioning. If you have a sub intimal cannulation this does put the vessel at risk for rupture during

balloon dilation. Small aortic diameters may occlude limbs particularly using modular devices. And most importantly, the outflow must be optimized using stents distally if needed in the iliac arteries, but even more importantly, assuring that you've

treated the femoral artery and outflow to the profunda. Despite these good results, endograft use for occlusive disease is off label use and therefor not reimbursed. In comparison to open stents, endograft use is expensive and may not be cost effective. There's no current studies looking

into the cost/benefit ratio. Thank you.

- Nothing to disclose. This is a 19 year old female who was referred to us with abdominal pain. The pain had lasted a year and a half. She had epigastric pain. She had microscopic hematuria. Work up was done.

Notice the compression of the renal vein. Notice a selective venogram, and you can see it refluxes down. Here's an IVUS that demonstrates the narrowing. So, as most of you know, Nutcracker's compression of the left renal vein between the SMA and the aorta,

or the aorta and the vertebral body. Open transposition of left renal vein has been the conventional surgical therapy for this. The head is up here, feet are down here. The retroperitoneum has been taken back and reflected on to the abdominal wall

and you see the dissection proceeding here. I have to admit that this is speed up to about 1 1/2 times so that we could squeeze this in in under five minutes. You see the dissection, how nice it's cut out. Remember this is magnified 10 times.

Here you see the SMA and here's the area of the compression. Here you see the dissection of the contralateral vein. Cava is over here in the middle. Care is taken to identify and ligate the lumbar veins. We used to put clips on them,

but occasionally we've had a clip that slipped and it's a lot easier simply just to tie them off and divide them. Here you see one of the lumbars being tied off. This is really an important point we've learned in doing these cases.

Control is obtained vessel loops. Small clips placed on them and you can see where we are here, you see cava. We always sort of ... Here you'll see the adrenal vein being divided. That always mobilization.

We usually suture ligate the part on the cava because of the amount of bleeding that would cause if the clip popped off. We always leave a small clamp in here in the belly before we ligate anything,

before we open anything, that's sort of a just in case. Here we are immobilizing and dividing gonadals, ovarian veins and you can see the renal vein is basically skeletonized over here.

Here's cava over here and up top, flipped in the air, is your SMA. Now we're going to open the vein and you'll see us open and close it, but I want you to look at some point, in about a few seconds, we're actually going to show

you the web inside the vein after we close the cavotomy where the vein was just dissected off. We've switched from Prolene to Gore-tex. Here is the actual web being pulled out. She had an actual web right there and here is the more distal cavotomy.

Again we've sort of moved away from Prolene because we're fearful of the Prolene snapping. Because one of the problems with the robot is you really don't realize how hard you're pulling sometimes because there's no tactile feedback as you're using the robot.

Here's that extra clamp that I was talking about. The "just in case" as we call it. Here's your completed transposition of the renal vein. Incisions for ports. It's feasible, it's safe, it's effective. She went home the next day and I saw here in follow up.

She is about two years out now with no recurrent symptoms and doing well. Thank you very much.

- Ladies and gentlemen, I would like to thank Professor Veith for his kind invitation. A minimally invasive carotid endarterectomy. I have nothing to disclose. Here you can see the same patient operating with the classic carotid endarterectomy with normal incision and on the other side,

you will see the patient, the same patient after the minimal incision carotid endarterectomy. So ladies and gentlemen, if one can safely perform carotid endarterectomy by minimal incision, let's do it routinely. The technique of minimal incision carotid endarterectomy.

The incision must be done over a carotid bifurcation. In slim patient, it is easy to determine the location just by the palpation. By routinely, I advise to mark bifurcation by using ultrasound. Reaching the artery by tissue separation

along the border of sternocleidomastoid muscle. Once the artery is visualized, apply the vessel loop on the external carotid artery. If it is needed, on the thyroid artery. Pulling the external carotid artery vessel loop up to the opposite side,

and releasing posterior part of bifurcation enables visualization and applying vessel loop on the common carotid artery, about 15 millimeter down the bifurcation. Pulling the external carotid artery vessel loop down into the opposite side reveals anterior and posterior

portion of internal carotid artery. What is the most important? The vessel loop on the internal carotid artery must be located above atherosclerotic plague. Temporary clamping of internal carotid artery for 30 seconds should show if the shunt is needed.

If there is no neurological signs, we continue pulling all vessel loops to elevate the artery to the level of the skin. Typically, longitudinal incision from common carotid artery to internal carotid artery is performed. The main important maneuver

that led to perform this operation correctly and safely, this is eversion-like movement. After arteriotomy, I squeeze the artery, internal carotid artery, usually on the level of the end of the atherosclerotic plague, usually using the forceps.

I make eversion-like movement. This led me easily and safely remove that atherosclerotic plague from the internal carotid artery. Always allow one two second backflow from internal carotid artery

to remove potential debris by the blood flow. The same, unclamping common carotid artery for a short period of time to remove potential debris from the proximal part. Should a shunt be indicated, it is easy and quick to insert.

As a first step, the shunt is inserted into internal carotid artery. It is necessary to slightly loosen internal carotid vessel loop. In the same way, I put the shunt into the common carotid artery if it is needed.

Continued suture usually close the arteriotomy. If the diameter of the internal carotid artery is smaller than two millimeter, artificial patch can be easily used. Redon drainage is always used. I make another small incision for the Redon drain

due to very, very small incision for endarterectomy. And continued suture usually closes the wound for good cosmetical effort. Here, you can see the operation step-by-step. What I will now emphasize this group of patient.

This is symptomatic patient with a very soft atherosclerotic plague. In this series, our experience. This is 165 patients allocated into two groups. 122 patients in the minimal incision carotid endarterectomy group,

and 43 patients in classic endarterectomy group. Patients randomly allocated. Here, you can see the results three months, up to three months results. I will like to emphasize there were no nerve injury. Hoarseness and shunt was used in 12%

in minimal incision carotid endarterectomy group. Here, you can see in the first and second column, the results up to September 2017. Third and fourth column, the results up to September 2018. Here you can see some examples. Here you will see some more examples.

Here you will see the scar that is after the operation. So nearly no limitation in neck movement, quick wound healing, short hospital stay, and perfect cosmetic effect. So to conclude, ladies and gentlemen, this is the low risk operation.

This is the operation of quick recovery. Precautions and contraindication, according to my experience seems to be same as for classic carotid endarterectomy. Of course, further study is required. Minimal incision, I also used during the

aortobifemoral and femoropopliteal operations. I hope to show it next year. Ladies and gentlemen, when I was a young surgeon, it was said that big surgeon, big incision. I'd rather suggest, good surgeon should try to make the smallest incision possible.

Data and presented technique will be published. Thank you very much for your attention.

- The title is Why Open Surgery is Still the Best Treatment for Juxta- and Pararenal AAAs in Good Risk Patients: Technical Tips. This slide demonstrates the trends of AAA repair in our university, my institution. After the introduction of EVAR in 2007 in Japan, more than half of AAA was treated with endovascular repair.

However, open repair also remains the mainstay. After the introduction of EVAR, comparing the percentage of open repair with renal artery clamping between pre and post EVAR, there was a significant increase. Recently EVAR with fenestrated graft and snorkel technique

has been performed for the treatment of JAAA in high risk patients with favorable outcome. However, open surgery is typically used as first-time treatment in low-risk patients. We demonstrated the outcome with results in Journal of Vascular Surgery in 2018.

Among 451 open surgery, 111 underwent repair of JRAA aortic aneurysms. In hospital death was less than 1%. This study suggests that open repair of juxtrarenal and pararenal aortic aneurysms can be done safely. We will show the technical tips for juxta-

and pararenal AAAs. Operative approach, manipulation of the left renal vein, method of anastomosis and method of reconstruction of the renal artery. The position of proximal clamps were decided based on the anatomy of the aneurysmal neck,

which was two approach. One was transperitoneal approach. Proximal clamp site, it's placed in inter-renal was suprarenal, which is proximal upwards. Like this 3DCT, if the orifice of the renal artery and the SMA are at the same level,

we choose supra-celiac clamping. In those cases, we user a transabdominal approach with left-sided medial visceral rotation. As you can see the descending colon and pancreas, spleen and stomach are rotated anteriorly and medially, leaving the left kidney, adrenal gland, ureter

and gonadal vein in situ. Using this approach, we can control the orifice of the celiac artery and the SMA and both renal arteries and therefore we can directly clamp all of these arteries. After exposure, the proximal anastomosis was made without PCPS.

After finishing the proximal anastomosis, the clamp was then moved down to the graft to perfuse the renal arteries. The next, manipulation of left renal vein. This is a dissection of renal vein and dissection of the branches

such as the adrenal vein or gonadal vein frees the left renal vein and makes it easier to expose the proximal aorta and to perform proximal anastomosis. The next is on the left side with the division and stump closure.

Divide the renal vein proximally to the adrenal vein and gonadal vein and they close the stump by 4-0 prolene running sutures. On the left side it's a very rare case, but sometimes re-anastomosis in the vein. We demonstrated renal artery clamping and left renal vein

division during abdominal aortic aneurysm repair. That's not in itself compromised for long time renal function. In reaction to that, we demonstrate that in publication in Journal of Vascular Surgery, the division had no significant impact

on CRD for up to two years during follow-up. Then this is initially pressed on posterior and ends on anastomosis was performed by placing a 3-0 Prolene suture in the posterior wall and continued anteriorly in a running fashion. Regarding the renal artery perfusion,

perfusion it with cold ringer solution undertaken. The bypass graft for the renal artery was anastomosed to the main body or limb of the main graft in advance, then the main body was anastomosed to the aorta. During the aorta clamping, the renal artery was perfused with chilled solution.

Finally we demonstrated the technical tips for juxta- and pararenal AAAs. Thank you for your attention.

- Thank you Dr. Veith for this opportunity again, and, like to show you that I have no disclosures relevant to this talk. The objective is to report the management and outcome of five cases of brachial artery injury in children with supracondylar humerus fractures at our institution over the last few years,

and then emphasize the importance of close observation and low threshold for surgical exploration in these cases. The classification of supracondylar fractures is on the Gartland system, and typically the vascular injuries are associated with Type 3, although there are some reported cases with Type 2.

Supracondylar humerus fractures make up about 70% of elbow fractures in children. Displacement and deformity can injure the median nerve, as well as the brachial artery. And up to 20% of children will present with an abnormal vascular exam, on initial evaluation.

There is no doubt what you do for the ischemic hand, is the exploration of the brachial artery. However, for the perfused, pulseless hand, there is considerable controversy as to what one should do. If this is not recognized, and not appropriately treated, there can be significant complications,

which can affect the child for the rest of his life. Physical examination, including neurovascular examination is crucial. These are high-litigation cases, and just writing on your record that neurovascular status is intact, is totally inadequate.

With reference to this particular fracture, evaluation for median nerve intactness, and function of the anterior interosseous nerve in particular, is very important, as I'll show you in just a slide, where they can be associated with arterial injuries. Ladies and gentlemen, this is why

you have the pink pulseless hand, despite obstruction or interruption of the brachial artery, going to these rich collaterals around the elbow. The hand can still be pink, and pulseless. This is a demonstration of the coexisting injury when you have median nerve and brachial artery

damaged by the anteromedial location. This location of the proximal fracture fragment. And many have suggested routine vascular exploration for this sort of injury. The most common finding that we find when we explore the brachial artery

with supracondylar fractures of the humerus, is the artery is tethered between the fracture fragments. This is yet another example, this is the brachialis sign, where the proximal fragment can buttonhole through the brachialis muscle. Most open fractures will need brachial artery exploration

at the time of reduction of the fracture. So, now I would like to share with you these five cases that I mentioned, at Inova Fairfax Hospital. The average age was 5.4 years, and four of them were male, one was a female, and I described to you my personal experience

in taking care of these patients at the hospital, and then following them closely afterwards. Case one was a perfused hand, a pink perfused hand, without a pulse. And this gentleman, this patient presented the next day with compartment syndrome.

On exploration we found a tethered artery, we released it, patient has normal function at two years. Case two, had a positive pulse, positive Doppler signal, nothing was done, other than reducing the fracture, patient sent home, he represented with severe pain, and was found to have compartment syndrome on day three.

On exploration, the artery was tethered. It was released, no thrombectomy was necessary. Patient has been left with slight deficit in two fingers. Third case, perfused pulse, with no pulse was observed, and the last pulse the next day duplex showed that the brachial artery was obstructed.

It was transected, had a vein interposition, I used the basilic vein, and did thrombectomy, and normal function at four months. Fourth case, there was no pulse, no Doppler signal, immediate exploration, tethered artery, no thrombectomy, normal function restored.

Case five had a normal exam, but lost signal the next day, was found to have a massive hematoma. We evacuated the hematoma, normal function. Based on this, the treatment algorithm is when the patient has a positive pulse, has a palpable pulse,

we obviously would do nothing. When it's pulseless and ischemic, immediate surgical exploration. When it's perfused and there's no radial signal, diminished flow, on duplex ultrasound, we explore surgically,

and when there's a positive radial pulse, we observe for 24 to 48 hours before discharge. I have found pulse oximetry, in addition with duplex ultrasound, to be very helpful in this regard. And ladies and gentlemen, in conclusion,

immediate surgical exploration is mandated for the ischemic hand. We recommend close observation after reduction, despite return of palpable pulse or Doppler signal, due to risk of delayed ischemia or compartment syndrome, especially in young children.

Based on our experience, perfused pulseless hand is a consequence of arterial injury or spasm. And, if you use duplex ultrasound, as if we had done, we may have been able to avoid delayed care in three out of the five cases. We recommend immediate exploration, obviously for,

for absent pulse and ischemic hand. And we do recommend that early recognition of ischemia and compartment syndrome is paramount, and patient should be closely observed, even if they have a normal perfusion on reduction of the fracture site. Thank you so much.

- Few aspects of vascular surgery are more controversial than the management of neurogenic TOS as you heard earlier from Dr. Illig. I will propose that recurrent neurogenic TOS and persistent neurogenic TOS are even more of a challenge to deal with. Persistent neurogenic TOS

occurs in 10 to 15% of patients, and it's really the individual who fails to show any improvement following decompression. Recurrent, 15 to 30% of individuals who have some degree of partial or complete relief after the initial decompression.

This may be insidious or acute, and most often occurs somewhere within a two-year time frame. It could be a missed original diagnosis. Or it may be a secondary insult, a new stretch injury to the plexus,

incomplete rib resection, ectopic bands, scar tissue formation around the plexus, or pec minor syndrome. Pec minor syndrome is a frequent cause of recurrent and/or persistent neurogenic symptoms.

Its co-existence is over 50% of patients with neurogenic TOS. Sanders recognized in 2003 that pec minor causes the majority of recurrent neurogenic TOS symptoms. What is it?

Well the pec minor originates in the second, third, fourth, and fifth ribs, inserting on the coracoid process, which is part of the scapula. Muscle hypertrophy, spasm, and fibrosis will constrict those structures underneath,

which would be the axial artery vein and posterior to that will be your plexus. This was first described in '45 by Wright. Lord and Stone did five decompressions for what they defined as "hyperabduction syndrome." It was lost at some point,

rediscovered by Thomas then Doctor Sanders in 2004, who studied it prolifically, defined it as a sub-set of neurogenic TOS. There's a plethora of literature in this present day and age supporting pec minor syndrome.

Etiology: majority of people have trauma, most often in motor vehicle accident with a whiplash type of injury. Repetitive shoulder strain and spontaneous events can occur, inducing hyper-induction of the shoulder. Clinical symptoms are similar

between neurogenic and pec minor: weakness, pain in the neck, clavicular, anterior chest wall, trapezius region and then paresthesias, most often in the ulnar distribution. Isolated pec minor does happen. Typically these individuals

have fewer head and neck symptoms. And you'll notice the intensity of symptoms are significantly less. On your examination it's your standard TOS examination. Unfortunately provocative maneuvers are not as rewarding. You'll find that with isolated pec minor,

these individuals have fewer head and neck symptoms, typically with rotation, tilt, scalene compression. There's nothing really to see. But they do have point tenderness over the infraclavicular region, directly on top of the pec minor.

And you'll notice that with contraction of the pec major, the symptoms will be minimized. Sanders noted the three most common findings with pec minor syndrome are: tenderness to palpation in the subcoracoid space, ULTT and EAST.

You'll notice that in some of these patients who do have persistent or recurrent symptoms, quite likely, pec minor tenderness was missed in the original diagnosis. A thorough history and physical certainly is beneficial. Unfortunately there's no test or exam

that is pathognomonic for this. Appropriate imaging should be completed to rule out other pathology. And then a selective pec minor muscle block not a plexus block, but a minor block, a muscle minor block,

should be considered. EMG testing has mixed results. When you look at your muscle block, typically done with a baseline examination, you'll then inject lidocaine 4 cc with ultrasound guidance 45 degree angle to avoid dropping in the lung.

Inject in a two to three centimeter area. And what you're looking for is resolution of your tenderness, improvement of your symptoms, at rest and with provocative maneuvers. For those ones who have an unsuccessful block

you may want to consider repeating it, or consider performing a scalene block. For those individuals that do have pec minor syndrome, conservative therapy, which is range-of-motion and stretching, tends to work well, 50% resolution in eight-week window.

For those individuals who fail pec minor tenotomy, low-risk out-patient procedure, relatively quick recovery time. A variety of approaches have been defined in the literature. Here's one approach with a patient

previously undergoing neurogenic decompression with a superclavicular approach, this is the anterior axillary approach, with a two to three centimeter vertical incision in the anterior axillary line. The subcutaneous divide at the pec fascia

is open and the muscle is elevated with a Deaver retractor. You'll trace this up to the coracoid process, and with that being the case, you'll transect two to three centimeters of the pectoralis minor muscle,

and then release any fascial bands or accessory muscle around the neurovascular bundle. These patients have done well. With isolated tenotomy you'll see that there's an improvement anywhere from 90 to 100%. However, you need to take into consideration

a good number of these people have combined neurogenic TOS and pec minor syndrome. And with that being the case, the success may be a little bit less. So in summary, pec minor syndrome is a frequent cause

of recurrent or persistent neurogenic TOS. Isolated pec minor tends to have fewer head and neck symptoms. Diagnosis is aided with a block. And tenotomy appears to be safe and effective. Thank you.

- These are my disclosures. So aortic neck dilatation is not a new problem. It's been described even before the era of endovascular repair and it's estimated to occur in about 20% of all patients that undergo EVAR two years after the index procedure.

We're seeing more and more cases where patients that survive long enough after EVAR, they develop aortic neck dilatation beyond the nominal diameter of the endograft and like on this patient, this image, large type 1A endoleaks that are difficult to treat.

There's a number of factors that are contributing to aortic neck dilatation including a continuous outward force that is exerted by the endograft. Progression of aortic wall degeneration. Aneurisymal disease is a degenerative procedure.

The presence of endoleaks, particularly type two endoleaks have been implicated in aortic neck dilatation. And then incomplete seal at the proximal neck in the form of microleaks or positional leaks. HeliFX EndoAnchors as you heard were

designed to stabilize and improve the apposition of the endograft to the aortic neck. And as you saw on this video, their presence even when the super no fixation disengages from the wall of the aorta, may help stabilize the graft onto

the aorta and prevent type 1A endoleaks. About three or four years ago we started looking at the anchor registry data, trying to identify predictors of aortic neck dilatation in patients who are undergoing EVAR with EndoAnchors. We published those results about a year ago.

In terms of the one year mark, we had 267 patients in that cohort. We measured the aortic diameter at four different levels. 20 millimeters proximal to the lowest main renal artery and then at the level of the lowest renal artery, five and 10 millimeters distal to that.

We defined the change in diameter that occurred between the pre-implantation EVAR and the first post-implantation EVAR at about one month. As adoptive enlargement due mainly to the effect of endograaft and the interaction with the aortic wall.

And then we defined this dilatation, what occurred between the one month and the 12 month mark, post EVAR. We used 20 different variables and we ran all these variables at the three levels. And what we found in terms of

post-operative neck dilatation is that it occurred in 3.1% of patients at the level of the lowest renal artery. 7.7% five millimeters distal to it and 4.6% at 10 millimeters distal to it. And this is a dilatation with a threshold

of at least three millimeters. We felt that this was much more clinically relevant. In terms of protective factors for adaptive enlargement, the presence of calcium and the aortic diameter of the level of the lowest renal, both of these are easy to understand.

The stiffer the aorta, the lesser the degree of the immediate dilatation. But then when we looked at the true dilatation, we found out that the aortic neck diameter at the lowest renal artery was a significant risk factor as was Endograft oversizing.

So if you started with a large aorta to begin with, these patients were much more likely to develop neck dilatation and if you significantly oversize the endograft that was also an independent risk factor. On the other hand, the neck length as well as the number of EndoAnchors that

were placed in these patients, both appear to have independent protective effects. So the two year preliminary analysis results is what I'm going to present. The analysis is still ongoing, but now we have a larger number of patients, 674.

We performed the same measurements at the same levels. What we found in terms of time course and location of the aortic neck dilatation is that in the suprarenal site, there is negligible dilatation up to 24 months. The largest dilatation occurs at five millimeters,

but more interestingly, a significant number of patients did not even have endograft present in that location. And then at 10 millimeters distal to the lowest renal artery right where most of the aneurysm changes you would expect to occur,

that change in diameter was again negligible. Indirectly suggesting that EndoAnchors have protective effect. So these are our interesting, some interesting insights. Female sex and graft oversize do play a significant role in the post-operative neck dilatation.

With EndoAnchors implanted at the index procedure neck dilatation 10 millimeters distal to the lowest renal artery appears to be negligible both at 12 and at 24 months. But we're working to see a little bit more finer elements at this analysis.

As where exactly the EndoAnchors were placed and how this was associated with the changes in the aortic neck. We hope to have those results later this year. Thank you.

- Thank you very much for the privilege of participating in this iconic symposium. I have no disclosures pertinent to this presentation. The Atelier percutaneous endovascular repair for ruptured abdominal aortic aneurysms is a natural evolution of procedural technique due to the success of fully percutaneous endovascular

aortic aneurysm repair in elective cases. This past year, we had the opportunity to publish our data with regard to 30 day outcomes between percutaneous ruptured aneurysm repairs and surgical cutdown repairs utilizing the American College of Surgeons NSQIP database,

which is a targeted database which enrolls about 800 hospitals in the United States, looking at both the univariate and multivariate analyses comparing preoperative demographics, operative-specific variables and postoperative outcomes. There were 502 patients who underwent

ruptured abdominal aortic aneurysms that were included in this review, 129 that underwent percutaneous repair, whereas 373 underwent cutdown repair. As you can see, the majority were still being done by cutdown.

Over the four years, however, there was a gradual increase in the number of patients that were having percutaneous repair used as their primary modality of access, and in fact a more recent stasis has shown to increase up to 50%,

and there certainly was a learning curve during this period of time. Looking at the baseline characteristics of patients with ruptured aneurysms undergoing both modalities, there was not statistically significant difference

with regard to these baseline characteristics. Likewise, with size of the aneurysms, both were of equal sizes. There was no differences with regard to rupture having hypotension, proximal or distal extension of the aneurysms.

What is interesting, however, that the patients that underwent percutaneous repair tended to have regional anesthesia as their anesthesia of choice, rather than that of having a general. Also there was for some unexplained reason

a more significant conversion to open procedures in the percutaneous group as compared to the cutdown group. Looking at adjusted 30-day outcomes for ruptured endovascular aneurysm repairs, when looking at the 30-day mortality,

the operative time, wound complications, hospital length of stay, that was not statistically significant. However, over that four year period of time, there tended to be decreased hospital length of stay as well as decreased wound complications

over four years. So the summary of this study shows that there was an increased use of fully percutaneous access for endovascular repairs for ruptured aneurysms with noninferiority compared to traditional open femoral cutdown approaches.

There is a trending advantage over conventional surgical exposure with decreased access-related complications, as well as decreased hospital length of stay. Now, I'm going to go through some of the technical tips, and this is really going to be focused upon

the trainees in the room, and also perhaps those clinicians who do not do percutaneous access at this time. What's important, I find, is that the utility of duplex ultrasonography, and this is critical to delineate the common

femoral artery access anatomy. And what's important to find is the common femoral artery between the inguinal ligament and this bifurcation to the profunda femoral and superficial femoral arteries. So this is your target area. Once this target area is found,

especially in those patients presenting with ruptured aneurysm, local anesthesia is preferred over general anesthesia with permissive hypotension. This is a critical point that once you use ultrasound, that you'd want to orient your probe to be

90 degrees to the target area and measure the distance between the skin and the top of that artery. Now if you hold that needle at equidistance to that same distance between the skin and the artery and angle that needle at 45 degrees,

this will then allow you to have the proper trajectory to hit the target absolutely where you're imaging the vessel, and this becomes important so you're not off site. Once micropuncture technique is used, it's always a good idea just to use

a quick fluoroscopic imaging to show that your access is actually where you want it to be. If it's not, you can always re-stick the patient again. Once you have the access in place, what can then happen is do a quick angio to show in fact you have reached the target vessel.

This is the routine instructions for use by placing the percutaneous suture-mediated closure system at 45 degree angles from one another, 90 degrees from one another. Once the sheath is in place for ruptured aneurysm, the placement of a ballon occlusion

can be done utilizing a long, at least 12 French sheath so that they'll keep that balloon up in place. What's also good is to keep a neat operative field, and by doing so, you can keep all of these wires and sutures clean and out of the way and also color code the sutures so that you have

ease and ability to close them later. Finally, it's important to replace the dilator back in the sheath prior to having it removed. This is important just so that if there are problems with your percutaneous closure, you can always very quickly replace your sheath back in.

Again, we tend to color code the sutures so we can know which ones go with which. You can also place yet a third percutaneous access closure device if need be by keeping the guide wire in place. One other little trick that I actually learned

from Ben Starnes when visiting his facility is to utilize a Rumel mediated technique by placing a short piece of IV tubing cut length, running the suture through that, and using it like a Rumel, and that frees up your hand as you're closing up

the other side and final with closure. The contraindications to pREVAR. And I just want to conclude that there's increased use of fully percutaneous access for endovascular repair. There's trending advantages over conventional surgical exposure with decreased

access related complications, and improved outcomes can be attributed to increased user experience and comfort with percutaneous access, and this appears to be a viable first option. Thank you very much.

- Thank you very much (mumbling) and to the chairman. Good afternoon to everybody. We moved immediately to our topic. This is my disclosure. I would like to present you, introduce you, this new stent. They're a really MicroStent. 3.2 French system.

Very, very small, as you can see. And actually, they are available only for retrograde deployment. And very soon, we will have new devices for antegrade deployment. They are made with nitinol.

Two nitinols... Wires, sorry. My mind is, I'm getting older. Two nitinol wire, and they behave exactly as small supera. I can introduce you the case. That is a gentleman with a previous first toe amputation,

non-healing, and I previously recanalized the one month before posterior tibial artery, but this is a patient with a small artery disease. So, you can see here, the angio, this is a CO2 angio. We have a steel patent posterior tibial, but an occlusion, a long occlusion of the anterior tibial.

And you can see how poor is the vascularization in the foot. Of course, this time, the surgeon asked me to try to recanalize the anterior tibial artery, and for us, it became the new target. You can see how difficult is the progression of the wire in the middle of this black calcium

is a very severe calcificated patient. We achieve the rupture of the Asashi Astato 20 wire. So, it was fortunately, we didn't close the tip. We could remove and change in with an Abbott Command ES hook could fortunately, cross the lesion, but you can see here the balloon cannot advance completely

in this terrible calcified patient. So, we applied this technique. We (mumbling) the antegrade wire into a needle. You can see here the advancement of the wire out of the needle. And when we have in our hand the tip of the antegrade wire,

we can pull a little bit the wire in order to achieve the excess. And we can easily pull the system we have. So, after this phase, we can succeed in have a distal access. May I have the audio?

Yeah. There is no audio volume? Okay, so we achieve the retrograde. At full, that was the key. So, the balloon can come with the wire. But normally, does not work like this.

You have to do this. We put the (mumbling) in the stent (mumbling). So, we put the torca device in the proximal part of the wire very close to the cone of the balloon, the cone wire of the balloon so we can fix the torca device in the proximal part,

as you can see. And now we have one system. We have the wire fixed into the balloon, so if we pull the wire, we can pull the balloon. So, this is a system to advance in the severe calcified legion.

In the 90% of case, it work. Of course, if you fail with this system, I don't know how can we do. But after this manipulation, we could predilate successfully all the anterior tibial artery, and now we have the possibility to aggressively

prepare the vessel as we used to prepare the vessel for when we have severe calcification for a superior stent deployment in SFA. The same we used to do in the tibial vessel when we have this kind of calcification. So, I decided to predilate and dilate all the vessel

three millimeters, and the proximal part with aggressively, with a 3.5 millimeter. And you can see here, after the deployment, after the inflation of the balloon, the result was already good, but there was a residual stenosis at the osteum.

So, we repeated the inflation with a longer inflation with this 3.5 balloon, and the acute result, the acute remodel of the artery was really good. So, now the vessel is very well prepared, and if we think to the destiny of this kind of lesion,

we have to expect a restenosis or reocclusion probably due to the recoiling of the calcium, rather than a hyperplasia, or a myointimal hyperplasia. So, you can see here with a sheetless technique, we can insert from the distal access, this micro stent. It's really very clear.

It's very, very well built and projected, and so we can be very precise in the deployment of the proximal part, and you can see the pull out manipulation, but you can modulate the struts. You can pack when you need the struts in order to improve the resistance to the compression.

And you can see how good is the deployment of the stents in the proximal AT. Of course, the manipulation should be very gentle, and when we have the superposition of the two markers that the stent is completely deployed, and so we can achieve very good scaffold

all the lesion in this patient. So, after the deployment, we had a very nice acute result. Look at how good is the flow. And then we decided to continue interdistality in order to achieve some we can call outflow, which is very important.

But of course, this is a patient. We have Roberto in the panel who can be classified as a no-option patient, because the definition is we have clearly a small artery disease, and all the forefoot has no vessel. Look how difficult it was to achieve a reenter

into the dorsalis pedis after several perforation. This was my thought, but (laughs), it was really a war. At the end, you can see here the flow is exact, in the anterior, is exactly the same speed as in the posterior, but as you can see, in this patient,

the distribution system towards the forefoot is completely failed, so probably, the clinical improvement will be not so fine and not so good. But we before shift to the arterialization, of course, we tried to do our best.

You can see here again, the deployment, how clear is the deployment of the stent at the top at the proximal part of anterior tibial in another case. Very, very smooth after a very good preparation. So, again, the crucial key is to prepare very well the vessel in an aggressive way.

Our experience, we only performed seven cases. They're really very, very calcified. We lost some patient, but we checked three patients that are still patent with one occlusion. It's very encouraging and promising procedure. I really believe in this kind of scaffolding

when we have this kind of calcification. We are going to start a prospective study in order to collect more patient, of course. You can see here, this is one of the first patient perform with a very good result. And you can see here the flow in the doppler ultrasound.

So, thank you very much for your attention, and this is (speech drowned out by applause). - [Male] Thanks. Thanks, Marco. Fantastic, Ian. Any quick questions? Just a quick one.

I mean, in terms of distal preparation, can we use experience like supera with, as you say, slight over-dilating to allow it to sit unconstrained? - [Marco] Yeah, in effect, we have to try to oversize the vessel.

As Roberto used to say, the size is important, especially in below the knee. One of the main reasons of the failure of the global impact study was the undersizing of the angioplasty they perform. And when we have this kind of calcification,

of course, we have to try to crack the calcium in some way, and we have, of course, only balloon because it's difficult to think that an atherectomy device can cross this kind of lesion. So, we try to step-by-step to increase the size of the balloon and the pressure,

of course, in order to crack. And probably, probably could be one option for a better patency in this kind of patient. - [Male] Perfect. Just a quick question, Marco. Do you use a one-to-one ratio of this stent

with the (mumbling) vessel diameter? - [Marco] Yeah. - [Male] Or you use an-- - [Marco] Yes, in this case, we deploy the 3.5 millimeter stent after the 3.5 millimeter balloon angiopathy.

Of course, 3.5, it's a quite huge balloon for the proximal AT. Despite in some patient, we have to use this size of the balloon. - [Male] Okay, thanks, thanks very much, Marco.

- Lymphatic, so it's fun, actually, not to talk on venous interventions for once. And, naturally, the two systems are very different. But, on the other hand, they're also related in several ways and I will come back to that later. I have no disclosures, maybe only my gratitude to this man, Dr. Maxim Itkin,

who actually got me started in the field, and was gracious enough to supply me some of his material. And who is also responsible for making our lives way easier over the last years. Because in former times, we needed to do, to visualize the lymphatic system,

we needed to do pedal lymphangiography and that was very, very cumbersome. It took a long time and was very painful for the patient. And he introduced the ultrasound guided intranodal lymphangiography,

and that's fairly easy for most of us. With ultrasound you find a lymph node in the groin, you puncture that and you can control the needle position with contrast enhanced ultrasound and once you establish that position, you might do a MR lymphangiography.

Thereby showing, in this case, a beautiful, normal anatomy of the thoracic duct. I need to say, the variations in lymphatics are extreme. So, you can also visualize, naturally, the pathology, like for example, pulmonary lymphatic perfusion syndrome.

What's going on there. Normally, lymph courses up through thoracic duct, but in this case, you kind of have a reflux in the bronchial tree and lymph leakage. And you can image that again, beautifully with MR, which you can show extensive leakage

of lymph in the lung parenchyma. So you can treat that. How can you treat that? By embolization of the thoracic duct. But first we need to get into there, and that's not a very easy thing to do.

But now, again, with access to a lymph node in the groin, you can push lipiodol, and then visualize the cisterna chyli and access that transcutaneously with a 21/22 gauge needle and then push up a O-18 wire high up in the thoracic duct.

First you deploy some coils to prevent any leakage of glue inside the venous system, and then by microcatheter, you infuse glue all the way down, embolizing the thoracic duct. So, complete different group of lymphatic disorders is oriented in the liver and hepatic lymphatic disorders.

And maybe not everybody knows that, but 80% of the flow in the thoracic duct is caused by the liver and by the intestine. And many times in lymphatic disorders, there needs to be a combination of two factors. One factor is a venous variation of a,

sorry, an anatomical variation in lymph vessels and the other one is that we have an increase in lymph flow. And in the liver, that can be caused by a congestion of the liver, for example, cirrhosis, or a right side, that's congested heart failure.

What happens then is you increase the flow, the lymph flow, tremendously and if you also have a variation like in this case, when the vessels do not directly course towards the cisterna chyli, but in very close contact to the abdomen,

then you can have leakage of the lymph and leakage of proteins, which is a serious problem. So, what is then, to do next? You can access the lymph vessels in the liver by percutaneous access in the periportal space,

and induce some contrast and then later, visualize that one back, visualize that with dye that you can see with an endoscopy, thereby proving your diagnosis, and then, in a similar way,

you can induce lipiodol again with glue, embolizing the lymph vessels in the liver, treating the problem. In summary, popularity of lymphatic interventions really increased over the last years mainly because novel imaging,

novel interventional techniques, new approaches, and we all gained more experience. If you would like, I would guess that, we are at a phase where we were at venous, like 10, 15 years ago. If we are a little bit positive,

then the future is very bright. And within 10, 15 years, we find new indications and probably have much more to tell you. Thank you for your attention.

- Thank you very much, so my disclosures, I'm one of the co-PIs for national registry for ANARI. And clearly venous clot is different, requires different solutions for the arterial system. So this is a device that was built ground up to work in the venous system. And here's a case presentation of a 53 year old male,

with a history of spondylolisthesis had a lumbar inner body fusion, he had an anterior approach and corpectomy with application of an inner body cage. And you can see these devices here. And notably he had application of local bone graft and bone powder

and this is part of what happened to this patient. About seven days later he came in with significant left leg swelling and venous duplex showed clot right here, and this extended all the way down to the tibial vessels. And if you look at the CT

you can see extravasation of that bone powder and material obstructing the left iliac vein. And had severe leg swelling so the orthopedic people didn't want us to use TPA in this patient so we considered a mechanical solution. And so at this day and age I think goals of intervention

should be to maximize clot removal of course and minimize bleeding risk and reduce the treatment or infusion time and go to single session therapy whenever possible. Our ICUs are full all the time and so putting a lytic patient in there

reduces our ability to get other patients in. (mouse clicks So this is the ClotTriever thrombectomy device. It has a sheath that is a 13 French sheath and they're developing a 16 French, that opens up with a funnel

after it's inserted into the poplitiel. So the funnel is in the lower femoral vein and this helps funnel clot in when it's pulled down. The catheter has this coring element that abuts the vein wall and carves the thrombus off in a collecting bag

that extends up above to allow the thrombus to go into the bag as you pull it down. So you access the popliteal vein, cross the thrombosed segments with standard techniques and you need to then put an exchange length wire up into the SVC

or even out into the subclavian vein for stability. And then the catheter's inserted above the clot and is gradually pulled down, sort of milking that stuff off of the wall and into the bag that is then taken down to the funnel and out of the leg.

So this is the patient we had, we had thrombus in the femoral and up into the IVC. Extensive, you can see the hardware here. And it was very obstructed right at that segment where it was, had the bone material pushing on the vein it was quite difficult to get through there

but finally we did and we ballooned that to open a channel up large enough to accommodate ClotTriever catheter. We then did multiple passes and we extracted a large amount of thrombus. Some looking like typically acute stuff

and then some more dense material that may have been a few days worth of build up on the wall there. We then stinted with an 18 by 90 across the obstructed segment and this was our completion run.

It's not perfect but it looks like a pretty good channel going through. This is the hardware not obstruction at that level. Hospital course, the patient had significant improvement in their swelling by post-op day one. Was discharged on compression and anti-coagulation.

He returned about two months ago for his three month follow-up and really had very minimal symptoms in the left leg. Venous duplex showed that the left common femoral was partially compressible but did have phasic flow and the stent appeared to be open through it's course.

So of course this is an anecdote, this is early in the experience with this catheter. There have been numerous improvements made to ease the use of it and do it in fewer steps. And so we're starting a ClotTriever outcomes registry

to enroll up to 500 patients to begin to define outcomes with this device. It does offer the promise of single session therapy without lytic administration and we'll see how it performs and which patients it works best in through the registry.

Thank you very much.

- [Narrator] Good morning everyone. Again, thank you Dr. Veith for inviting me for this legendary meeting. I just love your meeting, thank you very much. Here you have my disclosures, I have said that the T-Branch device from Cook is not commercialized in the US

but it is in South America, now it's in Europe. Our presentation today is based in our article published German Vascular Therapy last year in August, Advanced technical considerations for implanting the T-branch off the shelf.

Branches stent-graft to treat Thoracoabdominal Aneurysms. I'm sure most of you already know this device. It's a off the shelf device from Cook. It has 202 millimeters in length. The proximal stent is 34 millimeters, the distal stent is 18 millimeters.

Of course, it has also four downward branch, so you have to adapt the anatomy of your patient and then to use this device in many situations. Here is a simple example, you can use this device in perirenal or superrenal aneurysms type 4. Just cutting one or two of the proximal stents.

Just be aware to (mumbles) the device in the (mumbles). So you can avoid the migration of the device. That's a good way to diminish the risks of paraplegia for you patient. The same way you can now cut the distal portion one or two stents, so and in cases you have

a previous device, you can pipe one in the leak, is we can show in cases, maxes lights, you can use this device. Also, the second component to anybody of the device, the bifurcated component can be cut. You can cut the proximal stents, you can cut

the distal stents, you can make that straight graft. So just like that, use it in many circumstances. And this is one of the maneuvers we use very often. We call that device driven by the sheath because you do a through and through wire and then you put that set the nose of our device

inside the sheath that come from the arm. So it helps by the avoid your device to touch the aort wall or even devices previously inserted. And also allow you to rotate the device to a correct position. Another maneuver is snare-ride technique that

we have already described in the Journal Endovascular Therapy last year. It's a very simple way so we can bring from the femoral access, we can bring the snare inside the one artery and that snare can capture a wire come from the arm, so we can

hold the position inside the target vessel. Here, an example that you can see all those maneuvers. This patient has a previous I-stent surgery and then the device that is probably the false lumen all the vessels come from this true lumen, which is secluded like capsule decortication.

They have minimal aortic communication. They've going to seen more details in the next slides. So here you can see the case that is a communication close to the celiac track and then is stuck. And then you have another communication, the intrarenal aort are very thin.

So here is a draw, you can see the first challenge was could we move a sheath, 12 branch sheath across the (mumbles) in the thin aorta and put that in the thin aorta, so without that, we could not do the case. We start the case doing that and as you can see,

we see that it was possible to do that, so we continue in the case. Following are challenges you will face was would we be able to cross this aort, very thin channel and to go there, to put the device here, and then to put the t-branch device to

all the branches from this true lumen. So here is our study, our plan was if you cross that communication, we put a t-branch here and used the celiac branch to TAAAs. Left renal artery, the celiac branch, the mysentary to branch the celiac artery, the left renal branch

to the mysentary artery and then right renal branch to right renal branch. So, that proves to be feasible. We could graft that communication and that adversary straight device to start the (cuts off). So here you see that the things

happened exactly as we planned it. The celiac was done by the SMA branch, the SMA done by the left renal, and the right renal by the right renal. At that point, we consider the game over. (cuts off) who could try the

celiac branch to the left renal. The angle was not preferable, so we come from the femoral artery in have access to left renal and open (mumbles) there and the diverse that wire should be put inside the left renal. Here you see the maneuver completed.

We advanced and hold the stent so we can have this branch also done. Here, you have a closed view of the left renal branch done by the celiac branch of the device. And now we have the final result of the branch done. How the bifurcate the device of completely

excluding the false movement of this complex dissection. So to illustrate this presentation, I bring you the control, one week control of this patient and could we fold the breasts where (mumbles) did it in the dissection, totally excluded from the circulation.

So, in conclusion ladies and gentlemen, I would say that the use of the branched stent-grafts in the treatment of Thoracoabdominal use is proven feasible, safe, and the off-the-shelf multibranched t-branch can be used in both urgent and elective scenarios.

Employing adjunctive maneuvers can increase the anatomic suitability of rience, these techniques have increased the applicability to 80 percent of the cases, included dissections of the small lumen.

I want to thank you all for your kind attention. Thank you, again, Frank for accepting my talk recorded. And I'm very pleased to answer questions by email or WhatsApp as you can see, this is live. Thank you very much.

- Thank you very much. I take over the presentation from Thomas Larzon, we, and different other people have the same approach to a ruptured triple A, trying to extend the advantages we have seen now, of an EVAR procedure in patients with inadequate anatomy, and to extend the limitation,

to patients with the less favorable anatomy. So, the concept of a ruptured EVAR has been already proven, with good research of three years, and I will build up, Thomas built up this presentation, on our so common experience that we published for fourteen years experience of two university centers,

performing EVAR on 100% of ruptured abdominal aortic aneurysms, over a 32 months period. So what we can see, is on the right side, this was the period where a part of the patient was treated by EVAR,

and the one that had not favorable anatomy were opened. On the left side, there is EVAR only, this a period 2009 to 11, you can see the effect of this change, is the operative cohort mortality moved from 26 to 24%, and total cohort mortality,

including to exclude the patient that are on feet, reduced from 33 to 27%. What changed also, is the protocol for anesthesia, so from a few patients that were treated under local anesthesia, actually, there are very few patients treated

just with general anesthesia primarily. What changed is the rejection rate, decreased from 10% to 4%, the age of the population treated increased, the part of women treated increased by 10%, and the amount of patients that are instable,

and treated, increased too. So, how to extend the limitation, the one is by using parallel grafts, or on table physician modified, extend graft to achieve what Benjamin does in his practice, a good seal proximal,

this is a three parallel graft, that worked very well. The other option, is to use Onyx for the distal landing zone, this is a technique that Thomas does use more liberally than we,

but is a good solution for patients where an IBD, for example, would not be possible, it doesn't require any special catheter, there is no contraindications due to tortuosity, and sealing is immediately obtained. Here, an example,

the aortoiliac, the main trunk, has been deployed here, then a (mumbles), the iliac extension is parked, can be deployed later, and as a Buddy catheter,

you can take a Bernstein catheter, you just position it in the origin of the hypogastric, or in the common iliac artery. Then, you deploy the distal extension, there is no more flow, slowly you'll stepwise,

5-10cc of Onyx can be applied, this allows to preserve the distal perfusion of the hypogastric, and to seal it. Sealing can also, with Onyx, can also be used in the proximal landing zone, there are two options,

here, the option with an instable patient that gets two parallel graphs with the remaining type 1 endoleak, you introduce your catheter through the leak, or the catheter inside the sack that is perfused, step wise, you will apply your Onyx.

Here, in another patient, of our experience, this is a suprarenal arteries after a triple A repair with EVAR that comes with the rupture, we combined here a chimney for the SMA, with a double brach device from Biotech,

deploy this, and you can see here there will be some leak. So, three days later, because the leak didn't have to do coagulation correct, once correct it didn't seal, we just very selectively, improvised with Onyx, the gap,

this is a three months outcome. Then, here a case of some Post EVAR with a type 1A endoleak, to extend this on the visceral aorta would have been very complex, this is why doctor Larson decided here just

to fill the whole sac with 60cc of Onyx, which worked very well. So, in Orebro, you can see that the 30-day mortality is 27%, the 90-day mortality is 30%, then the whole cohort,

including the 10% that have been excluded, has a mortality of 37 and 34%. From the different factor that was significant, you can see that local anesthesia works good, Aortic Balloon Occlusion works good, mortality in patients

with abdominal compartment syndrome is increased, mortality of patients in shock is increased, and finally, the mortality of patients having this adjunct procedure is not significantly increased, this holds true for the long-term outcomes.

So, we can see that by using adjuncts, every patient with a ruptured triple A can be offered an EVAR, eventually as a bridging procedure, chimney grafts can extend landing zones, Onyx can offer additional sealing options,

and valid long-term results for adjuncts has been proven. Thank you very much for your attention.

- So I have no disclosures related to this topic regarding open surgery. In terms of extent one, two, three thoracoabdominal aneurysms, Dr. Crawford's Benchmark Series really highlighted the risks of mortality in spinal cord ischemia related to this. And in terms of development techniques to improve,

these outcomes, most of them have centered on the spinal cord because the impact to spinal cord ischemia on perioperative mortality and longterm patient morbidity is so significant. Therefore, over the years we've developed things like CSF drainage, which has been studied in randomized

controlled trials, and have showed significant reduction in spinal cord ischemia rates. Early techniques also centered on improving spinal cord blood supply, such as a intercostal re-implantation techniques. However things like the collateral network concept

developed by Dr. Griepp suggest a routine sacrifice of these vessels, can be performed without major neurologic injury. Furthermore this elegant studies from Dr. Jacobs groups looking at MRAs after patient's thoracoabdominal aneurysms suggest that these collaterals actually originate caudal to the distal clamp

suggesting importance of pelvic perfusion. And therefore really sort of highlighting the importance of distal perfusion techniques during the operation. This has been shown by Dr. Safi's group. Again looking at adjunct's spinal cord drainage Motivo potentials and distal perfusion

with spinal cord ischemia rates are down to 2.4%. This has also been shown in Dr. Safi's hands to reduce organ disfunction, namely liver and kidney disfunction. As such, looking at this in the Medicare population in our own work looking at about 4,000 patients. Those that had extracorporeal circulation

utilized during their thoracobdominal repairs had a lower perioperative mortality and complications. And also improved survival. Based on these Dr. Cambria's experience at the Mass General Hospital, we sort of adopted a lot of these techniques.

And for the routine extent one through three, thoracoabdominal aneurysms we performed atrial femoral bypass, Motivo potential monitoring, inter-operative spinal drainage, and permissive hypothermia

resulting operative mortality rates of 4%, and paraplegia rates of 2.3%. However, in terms of the extent four thoracoabdominal aneurysms are approached just a little bit different. We don't use any of the aforementioned adjuncts. Namely in sort of a favor of a simplified technique,

whereas the thoracoabdominal incision is performed. We partially divide the diaphragm. Cold renal perfusion is used to protect the kidneys. A beveled proximal suture line facilitates a rapid revascularization of the visceral segment, and a routine left renal sidearm

to help reconstruct the left renal artery. This is resulted in excellent results. This is a paper I published in 2011, looking at about 178 patients with operative mortality rates of about 2.8%. Spinal cord ischemia rates of about 2.2%.

More recently, our resident Christopher Latz and partner Darrin Clouse have actually updated this data looking at about 226 patients. With again pretty good mortality and spinal cord ischemia rates. I do agree with Dr. Farber, that hospital volume

actually really is important. Overall mortality in the real world is 22% for thoracoabdominals. And this is outdated data, back in 2003, however high volume hospitals clearly perform better than low volume hospitals.

We've also looked at this in the Medicare population, looking at about 763 hospitals with 3,500 patients. High volume was defined at any hospital performing more than eight per year. With high volume hospitals having improved mortality. Furthermore Gib Upchurch recent work

suggested down-staging patients. Also improves the outcomes. And in this comparison of extent two thoracoabdominal aortic aneurysms where 25 were performed in stage fashion with a proximal TVAR followed by a lower thoracoabdominal, versus 88 standard.

There was lower major adverse events. Lower EBL, lower operative time, lower length of stay. Resulting in an operative mortality of about 4%. This is what a stage looks like with the TVAR on the left side, right side of the screen and the open reconstruction here.

Some terms of technical pearls to improve outcomes which is the focus of my talk, patient selection is very important. Anatomy and physiologic risk, spinal cord drainage, neuromonitoring, distal perfusion techniques, permissive hypothermia.

High volume centers, and down-staging to mitigate risks. So on to the debate. So Open versus Endo thoracoabdominal aneurysm prepare. This is a single, there center study looking at propensity match patients showing comparable results. Suggesting that both actually are good.

I think high volume centers that know how to do these operations are really the key in terms of improving outcome. And in the real world for using the French multi center experience, operative mortality and spinal cord ischemia rates

for branch and fenestrated graphs isn't as good as the IDE centers, which are obviously expert and have certain patient selections. Thank you.

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